Ebsteins anomaly of the tricuspid valve history and symptoms: Difference between revisions
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{{CMG}} and Claudia P. Hochberg, M.D. [mailto:chochber@bidmc.harvard.edu] | {{CMG}} and Claudia P. Hochberg, M.D. [mailto:chochber@bidmc.harvard.edu] | ||
'''Associate Editor-In-Chief:''' {{CZ}}}; [[Priyamvada Singh|Priyamvada Singh, MBBS]] [[mailto: | '''Associate Editor-In-Chief:''' {{CZ}}}; [[Priyamvada Singh|Priyamvada Singh, MBBS]] [[mailto:psingh13579@gmail.com]] '''Assistant Editor-In-Chief:''' [[Kristin Feeney|Kristin Feeney, B.S.]] [[mailto:kfeeney@elon.edu]] | ||
==Overview== | ==Overview== | ||
The symptoms | The common [[symptoms]] include [[cyanosis]], [[exertional dyspnea]], [[failure to grow]], [[fatigue]] and, [[palpitations]] may occur secondary to [[SVTs]] ([[supraventricular tachycardia]]) and [[WPW]] ([[Wolff-Parkinson-White syndrome]]), [[Paradoxical embolization]] may cause [[brain abscesses]] (right to left shunting due to interatrial communication). | ||
==History and symptoms== | |||
* [[ | The [[symptoms]] of Ebstein's anomaly depend upon the degree of apical displacement of the [[tricuspid valve]] leaflet as well as the degree of dysfunction of the [[tricuspid valve]]. If the [[tricuspid valve]] is severely deformed, [[fetal hydrops]] may occur. If the [[valve]] is functioning, [[patients]] may remain [[symptom]] free for many years.The common [[symptoms]] include:<ref name="pmid31384377">{{cite journal |vauthors=Holst KA, Connolly HM, Dearani JA |title=Ebstein's Anomaly |journal=Methodist Debakey Cardiovasc J |volume=15 |issue=2 |pages=138–144 |date=2019 |pmid=31384377 |pmc=6668741 |doi=10.14797/mdcj-15-2-138 |url=}}</ref> | ||
* [[ | *[[Cyanosis]] | ||
* [[ | *[[Exertional dyspnea]] | ||
*[[Failure to grow]] | |||
* [[Palpitations]] may occur secondary to SVTs ([[supraventricular tachycardia]]) and [[WPW]] ([[Wolff-Parkinson-White syndrome]]) | *[[Fatigue]] and [[cyanosis]] | ||
* [[Paradoxical embolization]] may cause [[brain abscesses]] (right to left shunting due to interatrial communication) | *[[Palpitations]] may occur secondary to [[SVTs]] ([[supraventricular tachycardia]]) and [[WPW]] ([[Wolff-Parkinson-White syndrome]]), | ||
*[[Paradoxical embolization]] may cause [[brain abscesses]] (right to left [[Shunt|shunting]] due to interatrial communication). | |||
==References== | ==References== | ||
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{{WS}} | {{WS}} | ||
[[Category: | [[Category:Cardiovascular system]] | ||
[[Category:Cardiology]] | [[Category:Cardiology]] | ||
[[Category:Congenital heart disease]] | |||
[[Category:Pediatrics]] | [[Category:Pediatrics]] | ||
[[Category:Disease]] |
Latest revision as of 20:56, 11 February 2020
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] and Claudia P. Hochberg, M.D. [2]
Associate Editor-In-Chief: Cafer Zorkun, M.D., Ph.D. [3]}; Priyamvada Singh, MBBS [[4]] Assistant Editor-In-Chief: Kristin Feeney, B.S. [[5]]
Overview
The common symptoms include cyanosis, exertional dyspnea, failure to grow, fatigue and, palpitations may occur secondary to SVTs (supraventricular tachycardia) and WPW (Wolff-Parkinson-White syndrome), Paradoxical embolization may cause brain abscesses (right to left shunting due to interatrial communication).
History and symptoms
The symptoms of Ebstein's anomaly depend upon the degree of apical displacement of the tricuspid valve leaflet as well as the degree of dysfunction of the tricuspid valve. If the tricuspid valve is severely deformed, fetal hydrops may occur. If the valve is functioning, patients may remain symptom free for many years.The common symptoms include:[1]
- Cyanosis
- Exertional dyspnea
- Failure to grow
- Fatigue and cyanosis
- Palpitations may occur secondary to SVTs (supraventricular tachycardia) and WPW (Wolff-Parkinson-White syndrome),
- Paradoxical embolization may cause brain abscesses (right to left shunting due to interatrial communication).
References
- ↑ Holst KA, Connolly HM, Dearani JA (2019). "Ebstein's Anomaly". Methodist Debakey Cardiovasc J. 15 (2): 138–144. doi:10.14797/mdcj-15-2-138. PMC 6668741 Check
|pmc=
value (help). PMID 31384377.