Wolff-Parkinson-White syndrome EKG

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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor-In-Chief: Cafer Zorkun, M.D., Ph.D. [2]

Overview

Wolff-Parkinson-White (WPW) pattern is characterized by the presence of characteristic ECG findings, such as a short PR interval and a delta wave. WPW syndrome is the occurrence of tachycardia with or without associated symptoms in a subject with existing WPW pattern.[1] Several types of arrhythmia can occur in WPW syndrome such as AV reentrant tachycardia (AVRT), atrial fibrillation,or atrial flutter, the most common of which is AVRT.[2] WPW syndrome can present as an orthodromic or antidromic AVRT during which the delta wave no longer appears. Atrial fibrillation in a patient with WPW should be suspected when there is ECG findings of an irregularly irregular rhythm and absent P waves suggestive of atrial fibrillation in the context of a heart rate higher than 220 beats per minute.

WPW Pattern

Determining the location of the accessory pathway

Check lead V1
Negative delta wave in V1 = right ventricle Positive delta wave im V1= left ventricle
Negative delta wave and QRS in II, III, AVF Left axis Inferior axis Negative delta wave and QRS in II, III, AVF Isoelectric or negative delta I, AVL, V5, V6
Posteroseptal Right free wall Anteroseptal Posteroseptal Lateral

WPW Syndrome

WPW syndrome is the occurrence of tachycardia with or without associated symptoms in a subject with existing WPW pattern.[3] Several types of arrhythmia can occur in WPW syndrome such as AV reentrant tachycardia (AVRT), atrial fibrillation,or atrial flutter, the most common of which is AVRT.[2]

Orthodromic AVRT

In orthodromic AVRT, the anterograde conduction (from the atrium to the ventricle) passes through the AV node and the retrograde conduction (from the ventricle to the atrium) passes through the accessory pathway. Orthodromic AVRT occurs in approximately 90 to 95% of WPW.
The EKG findings include:

Antidromic AVRT

The anterograde conduction (from the atrium to the ventricle) passes through the accessory pathway and the retrograde conduction (from the ventricle to the atrium) passes through the AV node. It apprears in less than 10% of WPW.
The EKG findings include:

Clinical Manifestations

  1. The most common form of paroxysmal tachycardia in these patients is a circus movement tachycardia (CMT) incorporating the AP.
  2. The CMT utilizes the following structures: the AV node, the His-Purkinje system, the ventricular myocardium (from the terminal portion of the His system to the ventricular end of the AP), the AP itself, and the atrial myocardium itself from the atrial insertion of the AP to the AV node.
  3. This circuit can conduct in both directions:
    • Type I A CMT:
      1. This is the usual form of the CMT in patients with WPW.
      2. Is antegrade through the AV node, VA conduction through the AP.
      3. The QRS complex during the tachycardia shows either normal intraventricular conduction or typical bundle branch block configuration.
      4. Symptoms: Palpitations (97%), dyspnea (57%), anginal pain (56%), perspiration (55%), fatigue (41%), anxiety (30%), dizziness (30%),polyuria (26%).
      5. This is also called orthodromic reentrant tachycardia.
      6. There is no delta wave.
      7. The rate is 140 to 250 bpm.
      8. It is faster than the rate of tachycardia due to reentry in the AV node.
      9. Often triggered by a PAC


    • Type I B CMT:
      1. Anterograde down the accessory pathway, retrograde in the AVN-His pathway.
      2. The QRS is widened (wide QRS)
      3. This form is rare.
      4. Also called antidromic reentrant tachycardia.


    • Type II CMT (intra-AV nodal):
      1. Anterograde pathway is an AV nodal slow pathway, the retrograde pathway is an AV nodal fast pathway.
      2. No evidence of ventricular pre-excitation during the tachycardia.
    • Type III CMT (uses two accessory pathways):
      1. Conducts anterograde down one accessory pathway and retrograde up a second accessory pathway.
      2. These patients can also experience atrial tachycardias and ventricular tachycardias.

Atrial Fibrillation in WPW

Atrial fibrillation in WPW syndrome is life-threatening because it might lead to ventricular tachycardia. The suggestive findings on the ECG include:

  • Irregularly irregular rhythm
  • Absent P wave
  • Ventricular rate >220 beats per minute

Examples

Shown below is an EKG of Wolff-Parkinson-White syndrome demonstrating slurred upstroke of the QRS complex (>110 milli sec), resulting in a delta-wave (arrow). The EKG also shows a short PR interval.

Copyleft image obtained courtesy of ECGpedia,http://en.ecgpedia.org/wiki/Main_Page


Shown below is an EKG showing a slurred upstroke QRS complex which is best appreciated in the precordial leads and a PR interval of less than 120 ms (short PR interval) suggesting WPW syndrome.

Copyleft image obtained courtesy of ECGpedia,http://en.ecgpedia.org/wiki/Main_Page


Shown below is an EKG demonstrating a delta wave in leads V2, I, aVL, with wide QRS complexes and left axis deviation suggesting WPW syndrome.

Copyleft image obtained courtesy of ECGpedia,http://en.ecgpedia.org/wiki/Main_Page


Shown below is an EKG showing a short PR interval of less than 120 ms, delta waves in leads I, aVF, aVL and chest leads with wide QRS complexes indicating WPW syndrome.

Copyleft image obtained courtesy of ECGpedia,http://en.ecgpedia.org/wiki/Main_Page


For more EKG examples of Wolff-Parkinson-White syndrome click here.

References

  1. "Wolff-Parkinson-White Syndrome and Accessory Pathways". Retrieved 1 April 2014.
  2. 2.0 2.1 "ACC/AHA/ESC Guidelines for the Management of Patients With Supraventricular Arrhythmias—Executive Summary". Retrieved 15 August 2013.
  3. "Wolff-Parkinson-White Syndrome and Accessory Pathways". Retrieved 1 April 2014.

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