Non-Polio enterovirus infections pathophysiology

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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: {Sujaya}}

Overview

Enteroviral diseases are more likely to be severe in the immunocompromised, including patients with diabetes, HIV, neoplasms, or post-transplant status.The cellular uptake of enteorviruses is mediated by receptor molecules such as, intracellular adhesion molecule-1 (ICAM-1), low-density lipoprotein receptor (LDL-R), and non-protein factors such as heparan sulfate and sialic acid. Incubation periods range from 12 hours to 5 days, with experimental volunteers reporting symptoms several hours after aritficial inoculation.

Pathophysiology[1][2][3][4]

Non-polio non-rhinovirus enteroviruses

Rhinoviruses

References

  1. Nikonov OS, Chernykh ES, Garber MB, Nikonova EY (2017). "Enteroviruses: Classification, Diseases They Cause, and Approaches to Development of Antiviral Drugs". Biochemistry (Mosc). 82 (13): 1615–1631. doi:10.1134/S0006297917130041. PMC 7087576 Check |pmc= value (help). PMID 29523062.
  2. Royston L, Tapparel C (2016). "Rhinoviruses and Respiratory Enteroviruses: Not as Simple as ABC". Viruses. 8 (1). doi:10.3390/v8010016. PMC 4728576. PMID 26761027.
  3. Huang HI, Shih SR (2015). "Neurotropic Enterovirus Infections in the Central Nervous System". Viruses. 7 (11): 6051–66. doi:10.3390/v7112920. PMC 4664993. PMID 26610549.
  4. Jacobs SE, Lamson DM, St George K, Walsh TJ (2013). "Human rhinoviruses". Clin Microbiol Rev. 26 (1): 135–62. doi:10.1128/CMR.00077-12. PMC 3553670. PMID 23297263.


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