Buerger's disease laboratory findings: Difference between revisions
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{{Buerger's disease}} | {{Buerger's disease}} | ||
{{CMG}};{{AE}}{{HM}} | |||
==Overview== | |||
There are no diagnostic laboratory findings associated with Buerger's disease. Lab testing is done to exclude other vasculitides, such as [[Systemic lupus erythematosus|lupus]], [[scleroderma]] and [[mixed connective tissue disease]]. | |||
==Laboratory Findings== | ==Laboratory Findings== | ||
*There are no diagnostic laboratory findings associated with Buerger's disease. | |||
*Laboratory testing is performed to exclude other pathologies, and include:<ref name="pmid10995867">{{cite journal |vauthors=Olin JW |title=Thromboangiitis obliterans (Buerger's disease) |journal=N. Engl. J. Med. |volume=343 |issue=12 |pages=864–9 |date=September 2000 |pmid=10995867 |doi=10.1056/NEJM200009213431207 |url=}}</ref><ref name="pmid12710840">{{cite journal |vauthors=Maslowski L, McBane R, Alexewicz P, Wysokinski WE |title=Antiphospholipid antibodies in thromboangiitis obliterans |journal=Vasc Med |volume=7 |issue=4 |pages=259–64 |date=2002 |pmid=12710840 |doi=10.1191/1358863x02vm452oa |url=}}</ref><ref name="pmid20421527">{{cite journal |vauthors=Piazza G, Creager MA |title=Thromboangiitis obliterans |journal=Circulation |volume=121 |issue=16 |pages=1858–61 |date=April 2010 |pmid=20421527 |pmc=2880529 |doi=10.1161/CIRCULATIONAHA.110.942383 |url=}}</ref> | |||
**Acute phase reactants that indicate an active inflammatory process: | |||
***[[Erythrocyte sedimentation rate]] | |||
***[[C-reactive protein]] | |||
**Immunologic panel to rule out autoimmune disease: | |||
***[[Antinuclear antibody]] | |||
***[[Rheumatoid factor]] | |||
***[[Anti-centromere antibodies|Anticentromere antibody]] | |||
***[[Antiphospholipid antibody]] | |||
***Serum [[creatinine]] | |||
***[[Complement]] | |||
***SCL-70 | |||
**To rule out hypercoagulability: | |||
***[[Complete blood count]] | |||
***[[Liver function]] test | |||
***[[Prothrombin time|PT]], [[Partial thromboplastin time|PTT]] and [[INR]] | |||
***[[Antiphospholipid antibodies]] | |||
***[[Anti-cardiolipin antibodies|Anticardiolipin antibodies]] | |||
***[[Protein C]] and [[Protein S|S]] | |||
***[[Antithrombin III]] | |||
***[[Factor V Leiden]] | |||
***[[Prothrombin gene mutation G20210A|Prothrombin gene mutation]] | |||
**Toxicology screen | |||
***[[Cocaine]] | |||
***[[Amphetamine]] | |||
***[[Cannabis]] | |||
**Others | |||
***[[Glucose test|Fasting glucose]] | |||
***[[Urinalysis|Urine analysis]] | |||
==References== | ==References== | ||
{{reflist|2}} | {{reflist|2}} | ||
{{WH}} | {{WH}} | ||
{{WS}} | {{WS}} | ||
[[Category:Cardiovascular diseases]] | [[Category:Cardiovascular diseases]] | ||
[[Category:Angiology]] | [[Category:Angiology]] | ||
[[Category:Cardiology]] | [[Category:Cardiology]] | ||
[[Category:Rheumatology]] | [[Category:Rheumatology]] | ||
[[Category:Up-To-Date]] | |||
Latest revision as of 18:54, 1 May 2018
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1];Associate Editor(s)-in-Chief: Hadeel Maksoud M.D.[2]
Overview
There are no diagnostic laboratory findings associated with Buerger's disease. Lab testing is done to exclude other vasculitides, such as lupus, scleroderma and mixed connective tissue disease.
Laboratory Findings
- There are no diagnostic laboratory findings associated with Buerger's disease.
- Laboratory testing is performed to exclude other pathologies, and include:[1][2][3]
- Acute phase reactants that indicate an active inflammatory process:
- Immunologic panel to rule out autoimmune disease:
- To rule out hypercoagulability:
- Toxicology screen
- Others
References
- ↑ Olin JW (September 2000). "Thromboangiitis obliterans (Buerger's disease)". N. Engl. J. Med. 343 (12): 864–9. doi:10.1056/NEJM200009213431207. PMID 10995867.
- ↑ Maslowski L, McBane R, Alexewicz P, Wysokinski WE (2002). "Antiphospholipid antibodies in thromboangiitis obliterans". Vasc Med. 7 (4): 259–64. doi:10.1191/1358863x02vm452oa. PMID 12710840.
- ↑ Piazza G, Creager MA (April 2010). "Thromboangiitis obliterans". Circulation. 121 (16): 1858–61. doi:10.1161/CIRCULATIONAHA.110.942383. PMC 2880529. PMID 20421527.