Hyperpathia
| Hyperpathia |
|
WikiDoc Resources for Hyperpathia |
|
Articles |
|---|
|
Most recent articles on Hyperpathia Most cited articles on Hyperpathia |
|
Media |
|
Powerpoint slides on Hyperpathia |
|
Evidence Based Medicine |
|
Clinical Trials |
|
Ongoing Trials on Hyperpathia at Clinical Trials.gov Clinical Trials on Hyperpathia at Google
|
|
Guidelines / Policies / Govt |
|
US National Guidelines Clearinghouse on Hyperpathia
|
|
Books |
|
News |
|
Commentary |
|
Definitions |
|
Patient Resources / Community |
|
Patient resources on Hyperpathia Discussion groups on Hyperpathia Patient Handouts on Hyperpathia Directions to Hospitals Treating Hyperpathia Risk calculators and risk factors for Hyperpathia
|
|
Healthcare Provider Resources |
|
Causes & Risk Factors for Hyperpathia |
|
Continuing Medical Education (CME) |
|
International |
|
|
|
Business |
|
Experimental / Informatics |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Hyperpathia is a clinical symptom of certain neurological disorders wherein the patient experiences a greatly exaggerated pain sensation to nociceptive stimuli. This can be contrasted from allodynia in which the patient feels painful sensations from stimuli which are not painful to normal individuals.
Mechanism
Hyperpathia describes the neuropathic pain which the pain threshold on one hand is elevated and the other hand is central hyperexcited whenever there is a loss of fibres. Hyperpathia is underlying the peripheral or central deafferentation when the afferent inputs are lost.[1] Hyperpathia only occurs on neuropathic pain patients with the loss of fibres.
The International Association of the Study of Pain’s (IASP) definition of hyperpathia is that: A painful syndrome characterized by an abnormally painful reaction to a stimulus, especially a repetitive stimulus, as well as an increased threshold. The definition also complies with a note which is: It may occur with allodynia, hyperesthesia, hyperalgesia, or dysesthesia. Faulty identification and localization of the stimulus, delay, radiating sensation, and after-sensation may be present, and the pain is often explosive in character. The changes in this note are the specification of allodynia and the inclusion of hyperalgesia explicitly. Previously hyperalgesia was implied, since hyperesthesia was mentioned in the previous note and hyperalgesia is a special case of hyperesthesia.[2]
References
- ↑ Jensen, T. S. (1996). Mechanisms of neuropathic pain. In J. N. Campbell (Ed.), Pain, 1996, an updated review. (pp. 77-86). Seattle: IASP Press
- ↑ (I.A.S.P, 1986). Pain Supplement 3: Classification of Chronic Pain, Descriptions of Chronic Pain Syndromes and Definitions of Pain Terms. Amsterdam: Elsevier.
Template:Skin and subcutaneous tissue symptoms and signs Template:Nervous and musculoskeletal system symptoms and signs Template:Urinary system symptoms and signs Template:Cognition, perception, emotional state and behaviour symptoms and signs Template:Speech and voice symptoms and signs Template:General symptoms and signs