Tricuspid atresia physical examination: Difference between revisions

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*Left ventricle dilation
*Left ventricle dilation


Symptoms
 
 
== Symptoms ==


----
----


Symptoms of tricuspid atresia in neonates may include the following:
Symptoms of tricuspid atresia in neonates may include the following:
Central cyanosis in mucous membranes and tongue
 
Poor feeding and growth retardation
----
Difficulty in breathing
 
Rapid heart beat
*Central cyanosis in mucous membranes and tongue
Rapid breathing
*Poor feeding and growth retardation
*Difficulty in breathing
*Rapid heartbeat
*Rapid breathing
Symptoms of longstanding cyanosis and hyperviscosity syndrome as a result of secondary erythrocytosis in older children include the following:
Symptoms of longstanding cyanosis and hyperviscosity syndrome as a result of secondary erythrocytosis in older children include the following:
Headache
 
Alter mentation
----
Faintness
 
Dizziness
*Headache
Visual disturbances
*Alter mentation
Paresthesia
*Faintness
Tinnitus
*Dizziness
Myalgia
*Visual disturbances
*Paresthesia
*Tinnitus
*Myalgia
 
 
Physical Examination
Physical Examination
Patients with pulmonary stenosis and closed PAD usually appear cyanotic after birth.
Physical examination may be remarkable for:
Normal pulses
Diminished right ventricle impulse
Thrill due to VSD or severe PS
Holosystolic murmured in LSB due to VSD
The continuous murmur of PDA, occasionally
Systolic ejection murmur in left upper sternal border due to PS
clubbing in older patients and unrepaired disease.
Patients with high pulmonary blood flow without stenosis in the pulmonary artery and with VSD are not cyanotic at birth.
Physical examination may be remarkable for symptoms and signs of overt heart failure:
Tachypnea
poor feeding
poor growth
Laboratory Findings
In cyanotic older patients laboratory finding may include:


Polycythemia due to secondary erythrocytosis and
----
Elevated prothrombin time and partial thromboplastin time
 
Decreased levels of factors 5,7,8,9: qualitative and quantitative
*Patients with pulmonary stenosis and closed PAD usually appear cyanotic after birth.
Platelet disorder
*Physical examination may be remarkable for:
Increased fibrinolysis and paradoxical thrombotic tendency
 
Proteinuria
**Normal pulses
Hyperuricemia
**Diminished right ventricle impulse
Renal failure
**Thrill due to VSD or severe PS
Uric acid nephrolithiasis
**Holosystolic murmured in LSB due to VSD
**The continuous murmur of PDA, occasionally
**Systolic ejection murmur in left upper sternal border due to PS
**clubbing in older patients and unrepaired disease.
 
*Patients with high pulmonary blood flow without stenotic pulmonary arteries and with VSD are not cyanotic at birth.
*Physical examination may be remarkable for symptoms and signs of overt heart failure:
**Tachypnea
**poor feeding
**poor growth
 





Revision as of 14:50, 21 August 2020

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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor-In-Chief: Keri Shafer, M.D. [2] Priyamvada Singh, MBBS[3] Assistant Editor-In-Chief: Kristin Feeney, B.S. [4]

Physical Examination

Diagnosis

Diagnostic Criteria


The diagnosis of tricuspid atresia is made when at least of the following three diagnostic criteria are met in echocardiography: Absent of color flow doppler in tricuspid valve region

  • ASD or PFO
  • Small right ventricle
  • Left atrium dilation
  • Left ventricle dilation


Symptoms


Symptoms of tricuspid atresia in neonates may include the following:


  • Central cyanosis in mucous membranes and tongue
  • Poor feeding and growth retardation
  • Difficulty in breathing
  • Rapid heartbeat
  • Rapid breathing

Symptoms of longstanding cyanosis and hyperviscosity syndrome as a result of secondary erythrocytosis in older children include the following:


  • Headache
  • Alter mentation
  • Faintness
  • Dizziness
  • Visual disturbances
  • Paresthesia
  • Tinnitus
  • Myalgia


Physical Examination


  • Patients with pulmonary stenosis and closed PAD usually appear cyanotic after birth.
  • Physical examination may be remarkable for:
    • Normal pulses
    • Diminished right ventricle impulse
    • Thrill due to VSD or severe PS
    • Holosystolic murmured in LSB due to VSD
    • The continuous murmur of PDA, occasionally
    • Systolic ejection murmur in left upper sternal border due to PS
    • clubbing in older patients and unrepaired disease.
  • Patients with high pulmonary blood flow without stenotic pulmonary arteries and with VSD are not cyanotic at birth.
  • Physical examination may be remarkable for symptoms and signs of overt heart failure:
    • Tachypnea
    • poor feeding
    • poor growth











Common physical examination findings associated with fibroadenoma include:[1]

Appearance of the Patient

Tricuspid atresia is a disease of infants. The child is growth retarded and appears cyanotic. The cyanosis is progressive.

Vital Signs

Skin

Bluish discoloration of the skin may be seen.

HEENT

Nasal flaring may be seen.

Neck

  • Prominent pulsation of the neck veins

Heart

  • The apical impulse is hyper-dynamic and shifted to the left.
  • A thrill can be felt on the left parasternal region of the chest.
  • Intensity of S1 may be increased.
  • Split S2 may be present.
  • A holosystolic crescendo - decrescendo murmur may be present signifying the flow of blood through a ventricular septal defect.
  • Rough, systolic murmur best heard in the third intercostal space close to the sternum.

Abdomen

  • Liver is enlarged and pulsations may be felt in the right upper quadrant of the abdomen.
  • Splenic enlargement

Extremities

References

  1. ASTLEY R, OLDHAM JS, PARSONS C (1953). "Congenital tricuspid atresia". Br Heart J. 15 (3): 287–97. doi:10.1136/hrt.15.3.287. PMC 479498. PMID 13059216.

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