Contrast induced nephropathy natural history, complications and prognosis: Difference between revisions
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==Overview== | |||
The reported incidence of CIN varies widely depending on the presence or absence of risk factors, CIN associated with a significantly higher risk of in-hospital and 1-year mortality, even in patients who do not need dialysis. | |||
==natural history, complications and prognosis== | |||
There is a complicated relationship between CIN, comorbidity, and mortality. Most patients who develop CIN do not die from renal failure rather than from either a preexisting nonrenal complication, or a procedural complication. | |||
Nonrenal complications include procedural cardiac complications (eg, Q-wave MI, [[coronary artery bypass graft]] [CABG], [[hypotension]], [[shock]]), vascular complications (eg, femoral bleeding, [[hematoma]], [[pseudoaneurysm]], [[stroke]]), and systemic complications (eg, [[acute respiratory distress syndrome]] [ARDS], [[pulmonary embolism]]). | |||
==References== | ==References== |
Revision as of 18:06, 11 September 2013
Contrast Induced Nephropathy Microchapters |
Differentiating Contrast induced nephropathy from other Diseases |
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Overview
The reported incidence of CIN varies widely depending on the presence or absence of risk factors, CIN associated with a significantly higher risk of in-hospital and 1-year mortality, even in patients who do not need dialysis.
natural history, complications and prognosis
There is a complicated relationship between CIN, comorbidity, and mortality. Most patients who develop CIN do not die from renal failure rather than from either a preexisting nonrenal complication, or a procedural complication.
Nonrenal complications include procedural cardiac complications (eg, Q-wave MI, coronary artery bypass graft [CABG], hypotension, shock), vascular complications (eg, femoral bleeding, hematoma, pseudoaneurysm, stroke), and systemic complications (eg, acute respiratory distress syndrome [ARDS], pulmonary embolism).