Supraventricular tachycardia electrocardiogram

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Differentiating Among the Different Types of Supraventricular Tachycardia

Differentiating Supraventricular Tachycardia from Ventricular Tachycardia

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Acute Treatment of SVT of Unknown Mechanism
Ongoing Management of SVT of Unknown Mechanism
Ongoing Management of IST
Acute Treatment of Suspected Focal Atrial Tachycardia
Acute Treatment of Multifocal Atria Tachycardia
Ongoing Management of Multifocal Atrial Tachycardia
Acute Treatment of AVNRT
Ongoing Management of AVNRT
Acute Treatment of Orthodromic AVRT
Ongoing Management of Orthodromic AVRT
Asymptomatic Patients With Pre-Excitation
Management of Symptomatic Patients With Manifest Accessory Pathways
Acute Treatment of Atrial Flutter
Ongoing Management of Atrial Flutter
Acute Treatment of Junctional Tachycardia
Ongoing Management of Junctional Tachycardia
Acute Treatment of SVT in ACHD Patients
Ongoing Management of SVT in ACHD Patients
Acute Treatment of SVT in Pregnant Patients
Acute Treatment and Ongoing Management of SVT in Older Population

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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]

Overview

Electrocardiogram

The EKG below is an interesting recording that shows a supraventricular tachycardia. The heart rate is around 185 bpm. It is somewhat unusual presentation for someone with angina. The arrhythmia terminated with adenosine which has a powerful cholinergic effect that blocks conduction through the AV node.

Copyleft image obtained courtesy of ECGpedia, http://en.ecgpedia.org/wiki/File:E345.jpg


Shown below is an example of an ECG showing tachycardia at a rate of 190/min with narrow QRS complexes indicating supraventricular tachycardia.

Copyleft image obtained courtesy of ECGpedia,


Shown below is the recording of the patient who goes from sinus rhythm to at first a wide complex tachycardia at about 130/min. The wide QRS though disappears after nine complexes and is replaced by narrow complexes at a slightly slower rate. No p wave activity is seen. This a supraventricular tachycardia with a form of abberancy. In this case we are probably seeing a rate dependent left bundle branch block or the effect of a left bundle branch block which persists for the nine complexes because of continued block in the left bundle from the depolarizations from the intact right bundle.


Shown below is an example of an ECG demonstrating a rapid heart rate at the rate of nearly 300 beats per minute indicating a paroxysmal spraventricular tachycardia.


Shown below is an example of an EKG showing a supraventricular tachycardia with group ventricular beating with clusters of regular rhythm at about 215/min. The regularity and group beating suggest that this is an organized rhythm and not atrial fibrillation. Look carefully at the interval between the 6th and 7th beats in lead II. Clearly atrial activity is seen at about 215/min. This is an interesting case where the diltiazem has slowed down the SVT which has allowed faster conduction down the A/V node and hence an increase in the ventricular rate.


Shown below is the recording shows the intiation of supraventricular tachycardia. There appears to be a p wave on the last part of the last sinus t wave suggesting that this may be an ectopic atrial tachycardia or possibly an atypical form of A/V nodal reentry where one sees the retrograde p wave before the QRS.

Sources

Copyleft images obtained courtesy of ECGpedia, http://en.ecgpedia.org/index.php?title=Special:NewFiles&offset=&limit=500

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