Tricuspid atresia medical therapy: Difference between revisions

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*The mainstay of therapy for the cyanotic neonate with severe[[ pulmonary stenosis]] and small sized [[VSD]] is using[[ prostaglandin E1]] (PGE1) for keeping patency of [[ductus arteriosis]].
*The mainstay of therapy for the cyanotic neonate with severe[[ pulmonary stenosis]] and small sized [[VSD]] is using[[ prostaglandin E1]] (PGE1) for keeping patency of [[ductus arteriosis]].
*The mainstay of therapy for [[heart failure]] symptoms is using [[diuretic]] for reduction of [[congestion ]]and then starting [[ ACEI]].
*The mainstay of therapy for [[heart failure]] symptoms is using [[diuretic]] for reduction of [[congestion ]]and then starting [[ ACEI]].
Surgery
Surgery is the mainstay of therapy for tricuspid atresia.
        In the first 8 weeks of life if there is severe [[cyanosis]] and [[pulmonary obstruction]] and normal positioning [[aorta]] and [[pulmonary artery]], making a shunt between systemic [[subclavian artery]] to the[[ pulmonary artery]] is necessary which is called [[Blalock -Taussig]](BT shunt).
        If the [[pulmonary artery]] comes from the [[left ventricle]] and is [[overflowed]], [[pulmonary artery]] banding is useful for lowering the [[pulmonary blood flow]].
        In older children, bi-direction [[Glenn shunt]] which is the connection between [[superior vena cava ]]to the[[ pulmonary artery]] will be done for transferring the blood to the [[pulmonary system]].
Fontan procedure is a conduit between the inferior vena cava and the pulmonary artery and transfers the systemic venous blood to pulmonary circulation in the age of 2-3 years old.
Endocarditis prophylaxis before every procedure is recommended.


==References==
==References==

Revision as of 16:12, 24 August 2020

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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor-In-Chief: Keri Shafer, M.D. [2] Priyamvada Singh, MBBS [3]; Assistant Editor-In-Chief: Kristin Feeney, B.S. [4]

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