Wide complex tachycardia history and symptoms: Difference between revisions
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==History and Symptoms== | ==History and Symptoms== | ||
=== History=== | ===History=== | ||
The patient should be asked about drugs that are associated with ventricular tachycardia and if there is a history of ischemic heart disease which would dramatically increase the odds that the rhythm is VT. Wide complex tachycardia will be due to [[VT]] in 80% of cases and will be due to [[VT]] in 98% of cases if there's a history of either [[acute MI]] or structural heart disease. Only 7% of patients with SVT will have had a prior myocardial infarction (MI). VT or an accelerated idioventricular rhythm can be seen following reperfusion in STEMI. Digoxin, antiarrhythmics, phenothiazines, TCAs, and pheochromocytoma may also cause VT. Recent procedures such as cardiac catheterization, DC countershock, repair of congenital lesions are all associated iwth VT. A family history of [[sudden cardiac death]], a history of a [[channelopathy]] associated with [[arrhythmias]], and the hereditary [[Long QT syndrome]], and [[Brugada syndrome]] are all associated with VT. | The patient should be asked about drugs that are associated with ventricular tachycardia and if there is a history of ischemic heart disease which would dramatically increase the odds that the rhythm is VT. Wide complex tachycardia will be due to [[VT]] in 80% of cases and will be due to [[VT]] in 98% of cases if there's a history of either [[acute MI]] or structural heart disease. Only 7% of patients with SVT will have had a prior myocardial infarction (MI). VT or an accelerated idioventricular rhythm can be seen following reperfusion in STEMI. Digoxin, antiarrhythmics, phenothiazines, TCAs, and pheochromocytoma may also cause VT. Recent procedures such as cardiac catheterization, DC countershock, repair of congenital lesions are all associated iwth VT. A family history of [[sudden cardiac death]], a history of a [[channelopathy]] associated with [[arrhythmias]], and the hereditary [[Long QT syndrome]], and [[Brugada syndrome]] are all associated with VT. | ||
===Symptoms === | ===Symptoms=== | ||
*[[Shortness of breath]] | *[[Shortness of breath]] | ||
*[[Syncope]] | *[[Syncope]] | ||
Revision as of 16:57, 7 February 2013
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
History and Symptoms
History
The patient should be asked about drugs that are associated with ventricular tachycardia and if there is a history of ischemic heart disease which would dramatically increase the odds that the rhythm is VT. Wide complex tachycardia will be due to VT in 80% of cases and will be due to VT in 98% of cases if there's a history of either acute MI or structural heart disease. Only 7% of patients with SVT will have had a prior myocardial infarction (MI). VT or an accelerated idioventricular rhythm can be seen following reperfusion in STEMI. Digoxin, antiarrhythmics, phenothiazines, TCAs, and pheochromocytoma may also cause VT. Recent procedures such as cardiac catheterization, DC countershock, repair of congenital lesions are all associated iwth VT. A family history of sudden cardiac death, a history of a channelopathy associated with arrhythmias, and the hereditary Long QT syndrome, and Brugada syndrome are all associated with VT.
Symptoms
- Shortness of breath
- Syncope
- Sudden cardiac death would suggest a diagnosis of ventricular tachycardia