Rapidly progressive glomerulonephritis risk factors: Difference between revisions

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{{Rapidly progressive glomerulonephritis}}
{{Rapidly progressive glomerulonephritis}}
{{CMG}}; {{AE}} {{AEL}}  
{{CMG}}; {{AE}} {{JSS}}


==Overview==
==Overview==
Common risk factors in the development of rapidly progressive glomerulonephritis may be occupational, environmental, [[genetic]], and [[viral]]. ibodies t
Common risk factors in the development of rapidly progressive glomerulonephritis may be occupational, environmental, [[genetic]], and [[viral]].


==Risk Factors==
==Risk Factors==
Line 10: Line 10:


=== Common risk factrors ===
=== Common risk factrors ===
* Pre existing renal disease
* Pre existing [[renal disease]].


* Presence of HLA-DRB1, HLA DP1, DQ and DRB4s
* Presence of [[HLA]]DRB1, HLA DP1, DQ and DRB4s<ref name="pmid15652778">{{cite journal| author=Jagiello P, Gross WL, Epplen JT| title=Complex genetics of Wegener granulomatosis. | journal=Autoimmun Rev | year= 2005 | volume= 4 | issue= 1 | pages= 42-7 | pmid=15652778 | doi=10.1016/j.autrev.2004.06.003 | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=15652778  }} </ref>
* Upper respiratory tract infection
 
* Sepsis
* [[Upper respiratory tract infection]]
* [[Sepsis]]
* Tobacco smoking
* Tobacco smoking
* IV drug abuse
* Intravenous drug abuse


=== Less common risk factors ===
=== Less common risk factors ===

Latest revision as of 19:32, 24 July 2018

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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Jogeet Singh Sekhon, M.D. [2]

Overview

Common risk factors in the development of rapidly progressive glomerulonephritis may be occupational, environmental, genetic, and viral.

Risk Factors

Common risk factors in the development of rapidly progressive glomerulonephritis may be occupational, environmental, genetic, and viral. [1][2]

Common risk factrors

  • Presence of HLADRB1, HLA DP1, DQ and DRB4s[3]

Less common risk factors

  • Cocaine use
  • Exposure to hydrocarbons( formaldehyde)
  • HIgh concenteration of FiO2

References

  1. Hellmark T, Segelmark M (2014). "Diagnosis and classification of Goodpasture's disease (anti-GBM)". J Autoimmun. 48-49: 108–12. doi:10.1016/j.jaut.2014.01.024. PMID 24456936.
  2. Bombassei GJ, Kaplan AA (1992). "The association between hydrocarbon exposure and anti-glomerular basement membrane antibody-mediated disease (Goodpasture's syndrome)". Am J Ind Med. 21 (2): 141–53. PMID 1536151.
  3. Jagiello P, Gross WL, Epplen JT (2005). "Complex genetics of Wegener granulomatosis". Autoimmun Rev. 4 (1): 42–7. doi:10.1016/j.autrev.2004.06.003. PMID 15652778.

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