Psittacosis pathophysiology: Difference between revisions

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==References==
==References==
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[[da:Psittacosis]]
[[de:Ornithose]]
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[[it:Psittacosi]]
[[ja:オウム病]]
[[no:Papegøyesyke]]
[[pt:Ornitose]]
[[ru:Орнитоз]]
[[simple:Psittacosis]]
[[fi:Papukaijakuume]]
[[sv:Papegojsjuka]]
[[pl:Ornitoza]]


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Revision as of 14:59, 7 August 2012

Psittacosis Microchapters

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Patient Information

Overview

Historical Perspective

Classification

Pathophysiology

Causes

Differentiating Psittacosis from other Diseases

Epidemiology and Demographics

Risk Factors

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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Aditya Govindavarjhulla, M.B.B.S. [2]

Overview

Pathophysiology

Transmission : The organism is shed in the nasal secretions and in the stool from infected birds and carriers. The organism can live for a long period of time outside the body by drying to form dust and infecting the susceptible hosts as they breathe. Oral contamination are especially significant in nest boxes. As a general rule, inhaled Chlamydia will cause severe disease, while ingested Chlamydia will tend to develop into carriers. Mouth to beak contact, handling of sick birds can cause infection.

Chlamydia psittaci gets lodged in respiratory epithelium. It then spreads to regional lymph nodes and causes secondary bacteremia. The incubation period is 5 - 21 days depending upon the immunocompetency of the individual. [1]

References

  1. Grayston JT, Thom DH (1991). "The chlamydial pneumonias". Curr Clin Top Infect Dis. 11: 1–18. PMID 1867764.


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