Mitral regurgitation history and symptoms: Difference between revisions

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{{Mitral regurgitation}}
{{Mitral regurgitation}}
{{CMG}}; {{AE}} {{CZ}}; [[Varun Kumar]], M.B.B.S.; [[Lakshmi Gopalakrishnan]], M.B.B.S.
{{CMG}}; {{AE}} {{CZ}}; [[Varun Kumar]], M.B.B.S.; [[Lakshmi Gopalakrishnan]], M.B.B.S.; {{Rim}}


==Overview==
==Overview==

Revision as of 18:42, 8 September 2014



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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Cafer Zorkun, M.D., Ph.D. [2]; Varun Kumar, M.B.B.S.; Lakshmi Gopalakrishnan, M.B.B.S.; Rim Halaby, M.D. [3]

Overview

Acute and decompensated mitral regurgitation is associated with symptoms of congestive heart failure including dyspnea, PND, orthopnea, and exercise intolerance. In chronic compensated mitral regurgitation there may be few symptoms.

History

Patients with suspected or confirmed MR should be inquired about the acuity of the symptoms. The knowledge of the onset of the symptoms helps in the classification of MR into acute and chronic.

In addition, the patient should be asked about the symptoms of diseases that might have lead to primary MR, such as recent ST elevation myocardial infarction, infective endocarditis, connective tissue disease, mitral valve prolapse, and rheumatic heart disease. In addition, the history of the patient should include information about coronary artery disease or any other conditions that might have caused heart failure in order to rule out secondary ischemic MR and functional MR respectively. Exposure to radiation to the chest should also be reported.

Symptoms

Acute or Decompensated Mitral Regurgitation

The symptoms associated with acute or decompensated mitral regurgitation are:

Cardiovascular collapse with shock (cardiogenic shock) may be seen in individuals with acute mitral regurgitation due to papillary muscle rupture or rupture of a chordae tendineae.

Chronic Compensated Mitral Regurgitation

Individuals with chronic compensated mitral regurgitation may be asymptomatic, with a normal exercise tolerance and no evidence of heart failure. These individuals may be sensitive to small shifts in their intravascular volume status, and are prone to develop volume overload (congestive heart failure).

References


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