Diphtheria pathophysiology: Difference between revisions
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==Mechanism== | ==Mechanism== | ||
[[Diphtheria toxin]] catalyzes the [[ADP-ribosylation]] of, and inactivates, the [[Eukaryotic elongation factors|elongation factor]] eEF-2. In this way, it acts to inhibit [[Translation (biology)|translation]] during eukaryotic protein synthesis. | [[Diphtheria toxin]] catalyzes the [[ADP-ribosylation]] of, and inactivates, the [[Eukaryotic elongation factors|elongation factor]] eEF-2. In this way, it acts to inhibit [[Translation (biology)|translation]] during eukaryotic protein synthesis. | ||
==References== | ==References== | ||
Revision as of 16:50, 1 October 2012
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Diphtheria Microchapters |
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Diagnosis |
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Treatment |
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Case Studies |
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Diphtheria pathophysiology On the Web |
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American Roentgen Ray Society Images of Diphtheria pathophysiology |
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Risk calculators and risk factors for Diphtheria pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Mechanism
Diphtheria toxin catalyzes the ADP-ribosylation of, and inactivates, the elongation factor eEF-2. In this way, it acts to inhibit translation during eukaryotic protein synthesis.