Accelerated idioventricular rhythm

Revision as of 19:40, 8 August 2012 by WikiBot (talk | contribs) (Bot: Automated text replacement (-{{SIB}} + & -{{EH}} + & -{{EJ}} + & -{{Editor Help}} + & -{{Editor Join}} +))
(diff) ← Older revision | Latest revision (diff) | Newer revision → (diff)
Jump to navigation Jump to search
Accelerated idioventricular rhythm
12 lead EKG shows a slow and wide complexes with intermittent narrow complex beats. The 5th and 10th beats are sinus node originated complexes (sinus rhythm) and close examination of these beats will give a clue to understand the cause of the wide complex rhythm. ST elevation indicates an acute myocardial infarction. The wide complex beats represent accelerated idioventricular rhythm which is usually seen following reperfusion in acute MI
MeSH D016170

WikiDoc Resources for Accelerated idioventricular rhythm

Articles

Most recent articles on Accelerated idioventricular rhythm

Most cited articles on Accelerated idioventricular rhythm

Review articles on Accelerated idioventricular rhythm

Articles on Accelerated idioventricular rhythm in N Eng J Med, Lancet, BMJ

Media

Powerpoint slides on Accelerated idioventricular rhythm

Images of Accelerated idioventricular rhythm

Photos of Accelerated idioventricular rhythm

Podcasts & MP3s on Accelerated idioventricular rhythm

Videos on Accelerated idioventricular rhythm

Evidence Based Medicine

Cochrane Collaboration on Accelerated idioventricular rhythm

Bandolier on Accelerated idioventricular rhythm

TRIP on Accelerated idioventricular rhythm

Clinical Trials

Ongoing Trials on Accelerated idioventricular rhythm at Clinical Trials.gov

Trial results on Accelerated idioventricular rhythm

Clinical Trials on Accelerated idioventricular rhythm at Google

Guidelines / Policies / Govt

US National Guidelines Clearinghouse on Accelerated idioventricular rhythm

NICE Guidance on Accelerated idioventricular rhythm

NHS PRODIGY Guidance

FDA on Accelerated idioventricular rhythm

CDC on Accelerated idioventricular rhythm

Books

Books on Accelerated idioventricular rhythm

News

Accelerated idioventricular rhythm in the news

Be alerted to news on Accelerated idioventricular rhythm

News trends on Accelerated idioventricular rhythm

Commentary

Blogs on Accelerated idioventricular rhythm

Definitions

Definitions of Accelerated idioventricular rhythm

Patient Resources / Community

Patient resources on Accelerated idioventricular rhythm

Discussion groups on Accelerated idioventricular rhythm

Patient Handouts on Accelerated idioventricular rhythm

Directions to Hospitals Treating Accelerated idioventricular rhythm

Risk calculators and risk factors for Accelerated idioventricular rhythm

Healthcare Provider Resources

Symptoms of Accelerated idioventricular rhythm

Causes & Risk Factors for Accelerated idioventricular rhythm

Diagnostic studies for Accelerated idioventricular rhythm

Treatment of Accelerated idioventricular rhythm

Continuing Medical Education (CME)

CME Programs on Accelerated idioventricular rhythm

International

Accelerated idioventricular rhythm en Espanol

Accelerated idioventricular rhythm en Francais

Business

Accelerated idioventricular rhythm in the Marketplace

Patents on Accelerated idioventricular rhythm

Experimental / Informatics

List of terms related to Accelerated idioventricular rhythm

Cardiology Network

Discuss Accelerated idioventricular rhythm further in the WikiDoc Cardiology Network
Adult Congenital
Biomarkers
Cardiac Rehabilitation
Congestive Heart Failure
CT Angiography
Echocardiography
Electrophysiology
Cardiology General
Genetics
Health Economics
Hypertension
Interventional Cardiology
MRI
Nuclear Cardiology
Peripheral Arterial Disease
Prevention
Public Policy
Pulmonary Embolism
Stable Angina
Valvular Heart Disease
Vascular Medicine

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]

Associate Editor-In-Chief: Cafer Zorkun, M.D., Ph.D. [2]


Overview

The rate of cardiac contraction is determined by the intrinsic rate of depolarisation of the cardiac cells. In normal hearts the sinoatrial node in the atria depolarises at a rate of 70 beats per minute. This suppresses the intrinsic depolarisation of the other parts of the heart.

The accelerated idioventricular rhythm occurs when depolarisation rate of a normally suppressed focus increases to above that of the "higher order" focuses (the sinoatrial node and the atrioventricular node). This most commonly occurs in the setting of a sinus bradycardia.[3]

Accelerated Idioventricular Rhythms are ectopic ventricular rhythms at rates between 40 bpm and 100 to 120 bpm. The ventricular origin of this rhythm can be demonstrated by the usual EKG criteria which include AV dissociation, fusion, and capture complexes.

The incidence of Accelerated Idioventricular Rhythms following acute MI is reported to be between 8 and 36%. This rhythm can also be seen in patients with primary myocardial disease, hypertensive, rheumatic, and congenital heart disease. Digoxin may cause of accelerated idioventricular rhythm.

EKG characteristics

  • Regular rhythm at a rate of 40 to 100-120 BPM.
  • QRS complexes are abnormal and wide.
  • The ventricular complexes are usually but not necessarily dissociated from the P waves.
  • Ventricular capture and fusion beats are common.

Differential Diagnosis

  1. Because of its slower rate it may resemble NSR. Look for numerous fusion beats. The term accelerated isorhythmic ventricular rhythm has been suggested.
  2. Must be distinguished from junctional tachycardia with preexisting IVCDs. But in these patients there are no fusion or capture beats.

Clinical Correlation

  1. Seen in both AMIs and IMIs.
  2. Commonly seen following reperfusion.
  3. Usually occurs during sinus bradycardia.
  4. May also be caused by digitalis.

AIVR appears similar to ventricular tachycardia but is benign and doesn't need any treatment.

References

  1. Hammill S. C. Electrocardiographic diagnoses: Criteria and definitions of abnormalities, Chapter 18, MAYO Clinic, Concise Textbook of Cardiology, 3rd edition, 2007 ISBN 0-8493-9057-5
  2. Engelen DJ, Gressin V, Krucoff MW, Theuns DA, Green C, Cheriex EC, Maison-Blanche P, Dassen WR, Wellens HJ, and Gorgels AP. Usefulness of frequent arrhythmias after epicardial recanalization in anterior wall acute myocardial infarction as a marker of cellular injury leading to poor recovery of left ventricular function. Am J Cardiol 2003 Nov 15; 92(10) 1143-9. PMID 14609586.
  3. Ilia R, Zahger D, Cafri C, Abu-Ful A, Weinstein JM, Yaroslavtsev S, Gilutz H, and Amit G. Predicting survival with reperfusion arrhythmias during primary percutaneous coronary intervention for acute myocardial infarction. Isr Med Assoc J 2007 Jan; 9(1) 21-3. PMID 17274350
  4. http://www.emedicine.com/med/topic12.htm
  5. Hampton, John (1997). The ECG Made Easy.

Additional resources

Examples



Template:WikiDoc Sources