Bronchitis pathophysiology: Difference between revisions
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==Overview== | ==Overview== | ||
[[Acute bronchitis]] is the inflammatory response of the bronchial epithelium to infections or irritants. The pathophysiological finding seen with acute bronchitis include: thickening, hyperemia and edema of the bronchial mucosa. This in turn decreases the bronchial mucociliary function. As a result of which the air passages become clogged by debris and causes copious mucus secretion, which causes the characteristic cough of bronchitis. | |||
==Pathophysiology== | ==Pathophysiology== | ||
Revision as of 22:16, 28 February 2012
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
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Bronchitis Main page |
Overview
Acute bronchitis is the inflammatory response of the bronchial epithelium to infections or irritants. The pathophysiological finding seen with acute bronchitis include: thickening, hyperemia and edema of the bronchial mucosa. This in turn decreases the bronchial mucociliary function. As a result of which the air passages become clogged by debris and causes copious mucus secretion, which causes the characteristic cough of bronchitis.
Pathophysiology
- Acute bronchitis is the inflammatory response of the bronchial epithelium to infections or irritants.
- Thickening of the bronchial and tracheal mucosa due to inflammation is also seen.
- The hyperemia and edema of the bronchial mucosa decreases the bronchial mucociliary function. As a result of which the air passages become clogged by debris and causes copious mucus secretion, which causes the characteristic cough of bronchitis.
- In mycoplasma pneumonia, bronchial irritation results from the attachment of the organism to the respiratory mucosa resulting in sloughing of affected cells.
- Bronchitis caused by influenza virus shows an epithelial-cell desquamation in association with the presence of a lymphocytic cellular infiltrate
References