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==Overview==
==Overview==
Effective measures for the secondary prevention of hypoaldosteronism include liberal salt intake of 4gm/day (to increase plasma sodium concentration), decreasing potassium intake  and avoidance of drugs that affects renin angiotensin aldosterone system (RAAS) such as ACE inhibitors, ARBs, potassium sparing diuretics and β-Adrenergic receptor blockers.
Effective measures for the [[secondary prevention]] of hypoaldosteronism include liberal [[salt]] intake of 4gm/day (to increase [[plasma]] [[sodium]] [[concentration]]), decreasing [[potassium]] intake  and avoidance of [[drugs]] that affects [[renin angiotensin aldosterone system]] ([[RAAS]]) such as [[ACE inhibitor|ACE inhibitors]], [[ARBs]], [[Potassium sparing diuretic|potassium sparing diuretics]] and [[Beta blockers|β-Adrenergic receptor blockers]].


==Secondary Prevention==
==Secondary Prevention==
*Effective measures for the secondary prevention of hypoaldosteronism include:
*Effective measures for the [[secondary prevention]] of hypoaldosteronism include:
**Low potassium intake
**Low [[potassium]] intake
**Salt intake of 4gm/day
**[[Salt]] intake of 4gm/day
**Avoid drugs affecting the renin angiotensin aldosterone system (RAAS) such as:
**Avoid [[drugs]] affecting the [[renin angiotensin aldosterone system]] ([[RAAS]]) such as:
***ACE inhibitors
***[[ACE inhibitor|ACE inhibitors]]
***Angiotensinogen receptor blocker  
***[[ARBs|Angiotensinogen receptor blocker (ARBs)]]
***Potassium sparing diuretics
***[[Potassium-sparing diuretic|Potassium sparing diuretics]]
***β-Adrenergic receptor blockers
***[[Beta blockers|β-Adrenergic receptor blockers]]


==References==
==References==

Revision as of 14:42, 22 August 2017

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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Akshun Kalia M.B.B.S.[2]

Overview

Effective measures for the secondary prevention of hypoaldosteronism include liberal salt intake of 4gm/day (to increase plasma sodium concentration), decreasing potassium intake and avoidance of drugs that affects renin angiotensin aldosterone system (RAAS) such as ACE inhibitors, ARBs, potassium sparing diuretics and β-Adrenergic receptor blockers.

Secondary Prevention

References

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