Hyponatremia risk factors: Difference between revisions
| Line 71: | Line 71: | ||
* SIAD: SIADH and gain of function mutation of v2 receptors | * SIAD: SIADH and gain of function mutation of v2 receptors | ||
* Chronic diseases cirrhosis, congestive heart failure | * Chronic diseases cirrhosis, congestive heart failure | ||
* Hospitalized patients those with pneumonia, persons, persons admitted to | * Hospitalized patients those with pneumonia, persons, persons admitted to Intensive care unite and patients receiving hypotonic fluid <ref>{{Cite journal | ||
| author = [[K. A. Neville]], [[C. F. Verge]], [[A. R. Rosenberg]], [[M. W. O'Meara]] & [[J. L. Walker]] | | author = [[K. A. Neville]], [[C. F. Verge]], [[A. R. Rosenberg]], [[M. W. O'Meara]] & [[J. L. Walker]] | ||
Revision as of 20:20, 6 May 2018
|
Hyponatremia Microchapters |
|
Diagnosis |
|---|
|
Treatment |
|
Case Studies |
|
Hyponatremia risk factors On the Web |
|
American Roentgen Ray Society Images of Hyponatremia risk factors |
|
Risk calculators and risk factors for Hyponatremia risk factors |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Saeedeh Kowsarnia M.D.[2]
Overview
There are no established risk factors for [disease name].
OR
The most potent risk factor in the development of [disease name] is [risk factor 1]. Other risk factors include [risk factor 2], [risk fac tor 3], and [risk factor 4].
OR
Common risk factors in the development of [disease name] include [risk factor 1], [risk factor 2], [risk factor 3], and [risk factor 4].
OR
Common risk factors in the development of [disease name] may be occupational, environmental, genetic, and viral.
Risk Factors
- strenuous exercise like marathon and triathlon runner [1]
- Institutionalize schizophrenic patients
- Excess intake of water with no protein intake (↓ urea excretion causes ↓water excretion) like potomania
- Severe kidney disease
- Diuretics especially thiazides (low body weight and hypokalemia increase the risk for thiazide associated hyponatremia) [2]
- Drugs with different mechanisms
- SIAD: SIADH and gain of function mutation of v2 receptors
- Chronic diseases cirrhosis, congestive heart failure
- Hospitalized patients those with pneumonia, persons, persons admitted to Intensive care unite and patients receiving hypotonic fluid [3]
References
- ↑ Christopher S. D. Almond, Andrew Y. Shin, Elizabeth B. Fortescue, Rebekah C. Mannix, David Wypij, Bryce A. Binstadt, Christine N. Duncan, David P. Olson, Ann E. Salerno, Jane W. Newburger & David S. Greenes (2005). "Hyponatremia among runners in the Boston Marathon". The New England journal of medicine. 352 (15): 1550–1556. doi:10.1056/NEJMoa043901. PMID 15829535. Unknown parameter
|month=ignored (help) - ↑ Alexander A. Leung, Adam Wright, Valeria Pazo, Andrew Karson & David W. Bates (2011). "Risk of thiazide-induced hyponatremia in patients with hypertension". The American journal of medicine. 124 (11): 1064–1072. doi:10.1016/j.amjmed.2011.06.031. PMID 22017784. Unknown parameter
|month=ignored (help) - ↑ K. A. Neville, C. F. Verge, A. R. Rosenberg, M. W. O'Meara & J. L. Walker (2006). "Isotonic is better than hypotonic saline for intravenous rehydration of children with gastroenteritis: a prospective randomised study". Archives of disease in childhood. 91 (3): 226–232. doi:10.1136/adc.2005.084103. PMID 16352625. Unknown parameter
|month=ignored (help)