Creutzfeldt-Jakob disease overview: Difference between revisions
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==Diagnosis== | ==Diagnosis== | ||
===Laboratory Findings=== | ===Laboratory Findings=== | ||
The diagnosis of CJD is suspected when there are typical clinical symptoms and signs such as rapidly progressing [[dementia]] with [[myoclonus]]. Analysis of CSF for [[14-3-3 protein]] is done to establish diagnosis. | The diagnosis of CJD is suspected when there are typical clinical symptoms and signs such as rapidly progressing [[dementia]] with [[myoclonus]]. Analysis of CSF for [[14-3-3 protein]] is done to establish diagnosis.<ref>{{Cite web | last = | first = | title = https://www.aan.com/Guidelines/home/GetGuidelineContent/567 | url = https://www.aan.com/Guidelines/home/GetGuidelineContent/567 | publisher = | date = | accessdate = 14 February 2014 }}</ref> | ||
==References== | ==References== | ||
Revision as of 15:36, 14 February 2014
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Creutzfeldt-Jakob disease Microchapters |
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Differentiating Creutzfeldt-Jakob disease from other Diseases |
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Creutzfeldt-Jakob disease is a very rare and incurable degenerative neurological disorder (brain disease) that is ultimately fatal. Among the types of transmissible spongiform encephalopathy found in humans, it is the most common.[1]
Diagnosis
Laboratory Findings
The diagnosis of CJD is suspected when there are typical clinical symptoms and signs such as rapidly progressing dementia with myoclonus. Analysis of CSF for 14-3-3 protein is done to establish diagnosis.[2]
References
- ↑ "http://www.cdc.gov/ncidod/dvrd/cjd/". Retrieved 14 February 2014. External link in
|title=(help) - ↑ "https://www.aan.com/Guidelines/home/GetGuidelineContent/567". Retrieved 14 February 2014. External link in
|title=(help)