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		<swivt:creationDate rdf:datatype="http://www.w3.org/2001/XMLSchema#dateTime">2026-07-31T07:52:12+00:00</swivt:creationDate>
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		<rdfs:label>WBR0309</rdfs:label>
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		<property:AnswerA rdf:datatype="http://www.w3.org/2001/XMLSchema#string">Takayasu arteritis</property:AnswerA>
		<property:AnswerAExp rdf:datatype="http://www.w3.org/2001/XMLSchema#string">Takayasu arteritis is a chronic inflammatory disease of the large arteries, such as the proximal aorta and its large branches. It is common in young women. Pathogenesis of Takayasu arteritis is characterized by granulomatous inflammation and fibrosis, especially in the medial layer of the arterial wall. Although a large number of patients with vasculitis do not show any increase in inflammatory markers, classically vasculitis is still considered a systemic inflammation with elevated acute phase reactants.</property:AnswerAExp>
		<property:AnswerB rdf:datatype="http://www.w3.org/2001/XMLSchema#string">Renal artery stenosis</property:AnswerB>
		<property:AnswerBExp rdf:datatype="http://www.w3.org/2001/XMLSchema#string">Renal artery stenosis is a common cause of secondary hypertension. It is an atherosclerotic condition that eventually leads to renal failure due to renal hypoxia and inadequate nutrition of renal parenchymal cells caused by the stenotic artery. Atherosclerosis in the renal artery is almost always observed in the proximal to mid portion; in contrast to FMD which mostly occupies the mid to distal regions.</property:AnswerBExp>
		<property:AnswerC rdf:datatype="http://www.w3.org/2001/XMLSchema#string">Fibromuscular dysplasia</property:AnswerC>
		<property:AnswerCExp rdf:datatype="http://www.w3.org/2001/XMLSchema#string">Although FMD may be an incidental finding, it may present with hypertension due to alteration in renal hemodynamics.</property:AnswerCExp>
		<property:AnswerD rdf:datatype="http://www.w3.org/2001/XMLSchema#string">Secondary hyperaldosteronism</property:AnswerD>
		<property:AnswerDExp rdf:datatype="http://www.w3.org/2001/XMLSchema#string">Although secondary hyperaldosteronism may be a cause of secondary hypertension, the patient's findings are not consistent with secondary hyperaldosteronism.</property:AnswerDExp>
		<property:AnswerE rdf:datatype="http://www.w3.org/2001/XMLSchema#string">Pheochromocytoma</property:AnswerE>
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		<property:WBRKeyword rdf:resource="&wiki;Fibromuscular_dysplasia"/>
		<property:WBRKeyword rdf:resource="&wiki;Renal_artery_stenosis"/>
		<property:WBRKeyword rdf:resource="&wiki;Angiogram"/>
		<property:WBRKeyword rdf:resource="&wiki;Renal_disease"/>
		<property:WBRKeyword rdf:resource="&wiki;String_of_beads_appearance"/>
		<property:WBRKeyword rdf:resource="&wiki;Renal_angiography"/>
		<swivt:wikiPageModificationDate rdf:datatype="http://www.w3.org/2001/XMLSchema#dateTime">2020-10-28T00:08:37Z</swivt:wikiPageModificationDate>
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