Sandbox:khurram: Difference between revisions

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{{CMG}}; {{AE}} {{MKA}}
{{CMG}}; {{AE}}  
==Overview==
The exact pathogenesis of [disease name] is not fully understood.
 
OR
 
It is thought that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
 
OR
 
[Pathogen name] is usually transmitted via the [transmission route] route to the human host.
 
OR
 
Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
 
OR
 
 
[Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
 
OR
 
The progression to [disease name] usually involves the [molecular pathway].
 
OR
 
The pathophysiology of [disease/malignancy] depends on the histological subtype.


==Pathophysiology==


==Overview==
===Pathogenesis===
*The exact pathogenesis of [disease name] is not fully understood.
OR
*It is thought that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
*[Pathogen name] is usually transmitted via the [transmission route] route to the human host.
*Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
*[Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
*The progression to [disease name] usually involves the [molecular pathway].
*The pathophysiology of [disease/malignancy] depends on the histological subtype.


Alcoholic liver disease may be classified according to histology and symptoms into different subtypes: reversible and irreversible.
==Genetics==
*[Disease name] is transmitted in [mode of genetic transmission] pattern.
*Genes involved in the pathogenesis of [disease name] include [gene1], [gene2], and [gene3].
*The development of [disease name] is the result of multiple genetic mutations.


==Classification==
==Associated Conditions==


*Alcoholic liver disease may be classified according to histology into different categories:<ref name="pmid22489260">{{cite journal |vauthors=Bruha R, Dvorak K, Petrtyl J |title=Alcoholic liver disease |journal=World J Hepatol |volume=4 |issue=3 |pages=81–90 |year=2012 |pmid=22489260 |pmc=3321494 |doi=10.4254/wjh.v4.i3.81 |url=}}</ref>
==Gross Pathology==
**Alcoholic steatosis - reversible; active alcohol abuse / metabolic syndrome
*On gross pathology, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].
**Alcoholic steatohepatitis - reversible; active alcohol abuse / metabolic syndrome
**Cirrhosis - irreversible; inflammation absent when abstinence from alcohol is observed


{{familytree/start |summary=Sample 1}}
==Microscopic Pathology==
{{familytree | | | | | | | | A01 |A01=Alcoholic Liver Disease}}
*On microscopic histopathological analysis, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].
{{familytree | | | |,|-|-|-|-|^|-|-|-|-|-|.| | | }}
{{familytree | | | B01 | | | | | | | | | B02 | | |B01=Reversible|B02=Irreversible}}
{{familytree | |,|-|^|-|.| | | | | | | | |!| }}
{{familytree | C01 | | C02 | | | | | | | C03 |C01=Alcoholic Steatosis|C02=Alcoholic Steatohepatitis|C03=Cirrhosis}}
{{familytree/end}}


==References==
==References==
{{Reflist|2}}
{{Reflist|2}}
{{WH}}
{{WS}}

Revision as of 16:45, 27 November 2017


Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief:

Overview

The exact pathogenesis of [disease name] is not fully understood.

OR

It is thought that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].

OR

[Pathogen name] is usually transmitted via the [transmission route] route to the human host.

OR

Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.

OR


[Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].

OR

The progression to [disease name] usually involves the [molecular pathway].

OR

The pathophysiology of [disease/malignancy] depends on the histological subtype.

Pathophysiology

Pathogenesis

  • The exact pathogenesis of [disease name] is not fully understood.

OR

  • It is thought that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
  • [Pathogen name] is usually transmitted via the [transmission route] route to the human host.
  • Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
  • [Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
  • The progression to [disease name] usually involves the [molecular pathway].
  • The pathophysiology of [disease/malignancy] depends on the histological subtype.

Genetics

  • [Disease name] is transmitted in [mode of genetic transmission] pattern.
  • Genes involved in the pathogenesis of [disease name] include [gene1], [gene2], and [gene3].
  • The development of [disease name] is the result of multiple genetic mutations.

Associated Conditions

Gross Pathology

  • On gross pathology, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].

Microscopic Pathology

  • On microscopic histopathological analysis, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].

References

Template:WH Template:WS