Impetigo pathophysiology: Difference between revisions
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==Pathophysiology== | ==Pathophysiology== | ||
===Bullous Impetigo=== | ===Bullous Impetigo=== | ||
The following are important aspects in the pathophysiology of bullous impetigo:<ref name="pmid27617460">{{cite journal| author=Cohen PR| title=Bullous impetigo and pregnancy: Case report and review of blistering conditions in pregnancy. | journal=Dermatol Online J | year= 2016 | volume= 22 | issue= 4 | pages= | pmid=27617460 | doi= | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=27617460 }} </ref> | The following are important aspects in the pathophysiology of bullous impetigo:<ref name="pmid27617460">{{cite journal| author=Cohen PR| title=Bullous impetigo and pregnancy: Case report and review of blistering conditions in pregnancy. | journal=Dermatol Online J | year= 2016 | volume= 22 | issue= 4 | pages= | pmid=27617460 | doi= | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=27617460 }} </ref><ref name="pmid27458596">{{cite journal| author=Duggal SD, Bharara T, Jena PP, Kumar A, Sharma A, Gur R et al.| title=Staphylococcal bullous impetigo in a neonate. | journal=World J Clin Cases | year= 2016 | volume= 4 | issue= 7 | pages= 191-4 | pmid=27458596 | doi=10.12998/wjcc.v4.i7.191 | pmc=4945591 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=27458596 }} </ref> | ||
*Bullous impetigo is caused by exfoliative toxins which arereleased by ''[[Staphylococcus aureus|Stapphylococcus aureus]]''. | *Bullous impetigo is caused by exfoliative toxins which arereleased by ''[[Staphylococcus aureus|Stapphylococcus aureus]]''. | ||
*The toxins are of two types, A and B, and lead to the production of bullae in the superficial layer of epidermis. | *The toxins are of two types, A and B, and lead to the production of bullae in the superficial layer of epidermis. | ||
Revision as of 19:08, 12 April 2017
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
The infection is spread by direct contact with lesions or with nasal carriers. The incubation period is 1–3 days. Dried streptococci in the air are not infectious to intact skin. Scratching may spread the lesions.
Pathophysiology
Bullous Impetigo
The following are important aspects in the pathophysiology of bullous impetigo:[1][2]
- Bullous impetigo is caused by exfoliative toxins which arereleased by Stapphylococcus aureus.
- The toxins are of two types, A and B, and lead to the production of bullae in the superficial layer of epidermis.
- These bullae are flaccid and can rupture easily.
References
- ↑ Cohen PR (2016). "Bullous impetigo and pregnancy: Case report and review of blistering conditions in pregnancy". Dermatol Online J. 22 (4). PMID 27617460.
- ↑ Duggal SD, Bharara T, Jena PP, Kumar A, Sharma A, Gur R; et al. (2016). "Staphylococcal bullous impetigo in a neonate". World J Clin Cases. 4 (7): 191–4. doi:10.12998/wjcc.v4.i7.191. PMC 4945591. PMID 27458596.