Mirizzi's syndrome pathophysiology: Difference between revisions
Line 20: | Line 20: | ||
'''Type IV lesions''' are complete destruction of the bile duct. <ref name="pmid2597969">{{cite journal |vauthors=Csendes A, Díaz JC, Burdiles P, Maluenda F, Nava O |title=Mirizzi syndrome and cholecystobiliary fistula: a unifying classification |journal=Br J Surg |volume=76 |issue=11 |pages=1139–43 |date=November 1989 |pmid=2597969 |doi=10.1002/bjs.1800761110 |url=}}</ref> | '''Type IV lesions''' are complete destruction of the bile duct. <ref name="pmid2597969">{{cite journal |vauthors=Csendes A, Díaz JC, Burdiles P, Maluenda F, Nava O |title=Mirizzi syndrome and cholecystobiliary fistula: a unifying classification |journal=Br J Surg |volume=76 |issue=11 |pages=1139–43 |date=November 1989 |pmid=2597969 |doi=10.1002/bjs.1800761110 |url=}}</ref> | ||
In 2008, Beltran et al described additional '''Type V lesions''' referring to presence of any of the above 4 types plus the formation of cholecystoenteric fistula. <ref name="pmid29369192">{{cite journal |vauthors=Chen H, Siwo EA, Khu M, Tian Y |title=Current trends in the management of Mirizzi Syndrome: A review of literature |journal=Medicine (Baltimore) |volume=97 |issue=4 |pages=e9691 |date=January 2018 |pmid=29369192 |pmc=5794376 |doi=10.1097/MD.0000000000009691 |url=}}</ref> <ref name="pmid23002333">{{cite journal |vauthors=Beltrán MA |title=Mirizzi syndrome: history, current knowledge and proposal of a simplified classification |journal=World J. Gastroenterol. |volume=18 |issue=34 |pages=4639–50 |date=September 2012 |pmid=23002333 |pmc=3442202 |doi=10.3748/wjg.v18.i34.4639 |url=}}</ref> | |||
==References== | ==References== | ||
{{Reflist|2}} | {{Reflist|2}} |
Revision as of 04:26, 23 July 2020
Mirizzi's syndrome Microchapters |
Diagnosis |
---|
Treatment |
Case Studies |
Mirizzi's syndrome pathophysiology On the Web |
American Roentgen Ray Society Images of Mirizzi's syndrome pathophysiology |
Risk calculators and risk factors for Mirizzi's syndrome pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Pathophysiology
Mirizzi’s syndrome is caused by gallstone impaction in the cystic duct or neck of gallbladder resulting in chronic inflammation that leads to the compression, necrosis and fibrosis of common bile duct. This can result in fistula formation into the adjacent structures like common bile duct. The obstruction of bile duct either by direct compression from gallstone or scar formation results in obstructive jaundice.
It can be divided into four types:
Type I lesions involve external compression of the common bile duct without any fistula formation.
Type II lesions involve cholecystobiliary fistula with erosion of less than one‐third of the circumference of the bile duct.
Type III lesions are fistula that involve up to two‐thirds of the duct circumference.
Type IV lesions are complete destruction of the bile duct. [1]
In 2008, Beltran et al described additional Type V lesions referring to presence of any of the above 4 types plus the formation of cholecystoenteric fistula. [2] [3]
References
- ↑ Csendes A, Díaz JC, Burdiles P, Maluenda F, Nava O (November 1989). "Mirizzi syndrome and cholecystobiliary fistula: a unifying classification". Br J Surg. 76 (11): 1139–43. doi:10.1002/bjs.1800761110. PMID 2597969.
- ↑ Chen H, Siwo EA, Khu M, Tian Y (January 2018). "Current trends in the management of Mirizzi Syndrome: A review of literature". Medicine (Baltimore). 97 (4): e9691. doi:10.1097/MD.0000000000009691. PMC 5794376. PMID 29369192.
- ↑ Beltrán MA (September 2012). "Mirizzi syndrome: history, current knowledge and proposal of a simplified classification". World J. Gastroenterol. 18 (34): 4639–50. doi:10.3748/wjg.v18.i34.4639. PMC 3442202. PMID 23002333.