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Spontaneous Coronary Artery Dissection Microchapters
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Overview
Historical Perspective
Classification
Pathophysiology
Causes
Differentiating Spontaneous coronary artery dissection from other Diseases
Epidemiology and Demographics
Risk Factors
Screening
Natural History, Complications and Prognosis
Diagnosis
Diagnostic Approach
History and Symptoms
Physical Examination
Laboratory Findings
Electrocardiogram
Angiography
CT
MRI
Echocardiography
Other Imaging Findings
Other Diagnostic Studies
Treatment
Treatment Approach
Medical Therapy
Percutaneous Coronary Intervention
Surgery
Primary Prevention
Secondary Prevention
Cost-Effectiveness of Therapy
Future or Investigational Therapies
Case Studies
Type 1 Case 1 Case 2 Case 3 Case 4 Case 5 Case 6 Case 7 Type 2A Case 1 Case 2 Case 3 Case 4 Case 5 Case 6 Type 2B Case 1 Case 2 Case 3 Case 4 Type 3 Case 1 Case 2 Case 3 Case 4 Case 5 Case 6

Algorithm for the Angiographic Diagnosis and Confirmation of Spontaneous Coronary Artery Dissection

Presence of features that raise a high clinical index of suspicion for SCAD? (click for details) ❑  Myocardial infarction in young women (especially age ≤50) ❑  Absence of traditional cardiovascular risk factors ❑  Little or no evidence of typical atherosclerotic lesions in coronary arteries ❑  Peripartum state ❑  History of fibromuscular dysplasia ❑  History of relevant connective tissue disorder or systemic inflammatory condition     ❑  Marfan's syndrome     ❑  Ehlers-Danlos syndrome Type 4     ❑  Loeys-Dietz syndrome     ❑  Cystic medial necrosis     ❑  Systemic lupus erythematosus     ❑  Crohn's disease     ❑  Ulcerative colitis     ❑  Polyarteritis nodosa     ❑  Sarcoidosis     ❑  Churg-Strauss syndrome     ❑  Wegener's granulomatosis     ❑  Rheumatoid arthritis     ❑  Giant cell arteritis

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