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Revision as of 19:00, 5 August 2020 by Mitra Chitsazan (talk | contribs)
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{{familytree | G01 | | G02 | | G03 | | G04 |G01=

❑ Classic triad of:

Hypotension
Elevated JVP
Clear Lungs

Kussmaul sign
Pulsus paradoxus
Tricuspid regurgitation murmur
Atrioventrcicular dissociation
❑ Vagal symptoms:

Bradycardia
Nausea
Vomiting
Diaphoresis
Pallor
|G02=

RV dilatation
❑ Depressed RV systolic function
RV wall akinesia or dyskinesia
RA enlargement
❑ Elevated pulmonary pressures
Pulmonary regurgitation
Tricuspid regurgitation
❑ Increased right atrial pressure

|G03=
Gold standard diagnostic modality

❑ In the majority of RVMI:

❑ The culprit artery: Proximal RCA

❑ Occasionally:

❑ The culprit artery: Left circumflex artery or left anterior descending artery
|G04=

❑ Hemodynamically significant RVMI:

❑ Increased RAP>10 mmHg
RAP to PCWP ratio >0.8 (normal<0.6)
RAP within 5 mmHg of the PCWP
❑ Reduced cardiac index
❑ Disproportionate elevation of right-sided filling pressures: Hallmark of RVMI

❑ In concomitant LV dysfunction:

RAP to PCWP ratio can change

❑ Additional hemodynamic changes:

❑ Prominent [[Y-descend of the RAP
❑ Drop of the systemic arterial pressure >10 mmHg with inspiration
❑ "Dip and plateau" morphology and equalization of the diastolic filling pressures
|}}
 
 
 
 
All patients with acute inferior wall myocardial infarction (ST elevation in leads II, III, aVF)
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Obtain right-sided precordial leads
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
>= 1mm ST elevation in lead V4R
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Highly suggestive of RVMI
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Physical examination
 
 
 
Hemodynamic study