Idiopathic pulmonary fibrosis pathophysiology: Difference between revisions

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(Replaced content with "__NOTOC__ {{Idiopathic pulmonary fibrosis }} {{CMG}}; {{AE}} {{AEL}} ==Overview== ==Pathophysiology== ==Genetics== ==Microscopic pathology== ==Gross pathology== ==Re...")
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==Overview==
==Overview==
==Pathophysiology==  
==Pathophysiology==
 
=== Normal lung tissue ===
* Lungs are composed normally of extracellular collagen which allows the lungs to exert their breathing efforts.
* Different collagen types in the lung include the following:<ref name="pmid1916105">{{cite journal| author=van der Rest M, Garrone R| title=Collagen family of proteins. | journal=FASEB J | year= 1991 | volume= 5 | issue= 13 | pages= 2814-23 | pmid=1916105 | doi= | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=1916105  }}</ref>
** Type 1 and type 3 compose most of the lung tissue
** Type 2 is the main component of the cartilage of the main bronchi
** Type 4 forms the basement membrane
** Type 5 forms the interstitial tissue 
 
==Genetics==  
==Genetics==  
==Microscopic pathology==  
==Microscopic pathology==  
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[[Category:Pulmonology]]
[[Category:Pulmonology]]
[[Category:Disease]]
[[Category:Disease]]

Revision as of 15:07, 14 February 2018

Idiopathic pulmonary fibrosis Microchapters

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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Ahmed Elsaiey, MBBCH [2]

Overview

Pathophysiology

Normal lung tissue

  • Lungs are composed normally of extracellular collagen which allows the lungs to exert their breathing efforts.
  • Different collagen types in the lung include the following:[1]
    • Type 1 and type 3 compose most of the lung tissue
    • Type 2 is the main component of the cartilage of the main bronchi
    • Type 4 forms the basement membrane
    • Type 5 forms the interstitial tissue

Genetics

Microscopic pathology

Gross pathology

References

  1. van der Rest M, Garrone R (1991). "Collagen family of proteins". FASEB J. 5 (13): 2814–23. PMID 1916105.

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