Dyspepsia classification: Difference between revisions
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__NOTOC__ | __NOTOC__ | ||
{{Dyspepsia}} | {{Dyspepsia}} | ||
{{CMG}} | {{CMG}} {{AE}} {{Ajay}} | ||
==Overview== | ==Overview== | ||
Dyspepsia is broadly classified into | Dyspepsia is broadly classified into ulcer and non-ulcer dyspepsia. The latter is also known as functional dyspepsia. | ||
==Classification== | ==Classification== | ||
Dyspepsia may be broadly classified into two major sub-types: | |||
===Non-Ulcer | ===Ulcer dyspepsia=== | ||
Non-ulcer dyspepsia, also called functional dyspepsia (FD), is defined by the Rome III criteria as symptoms of epigastric pain or discomfort ( | * Ulcer dyspepsia accounts for 20-30 % of all dyspepsia cases and is caused by [[peptic ulcer disease]] and [[gastroesophageal reflux disease]] [[GERD|(GERD]]). | ||
*Gastric motor function | * Structural disease of the [[gastrointestinal tract]] exists in ulcer dyspepsia and hence [[endoscopy]] is abnormal. | ||
*Visceral sensitivity | |||
*Helicobacter pylori infection | ===Non-Ulcer dyspepsia=== | ||
*Psychosocial factors | * Non-[[ulcer]] dyspepsia, also called functional dyspepsia (FD), is defined by the [[Rome III criteria]] as [[symptoms]] of [[epigastric pain]] or [[discomfort]] (found in 89-90% of cases), [[postprandial]] fullness (75-88%), and early [[satiety]] (50-82%) within the last 3 months with [[symptom]] onset at least 6 months earlier. | ||
* Patients should not have any evidence of structural disease and predominant [[symptoms]] of [[gastroesophageal reflux]] are exclusionary.<ref>{{cite journal |author=Lacy BE, Talley NJ, Locke GR, ''et al.'' |title=Review article: current treatment options and management of functional dyspepsia |journal=Aliment. Pharmacol. Ther. |volume=36 |issue=1 |pages=3–15 |year=2012 |month=July |pmid=22591037 |doi=10.1111/j.1365-2036.2012.05128.x |url=}}</ref> | |||
* Causes of functional dyspepsia are not clear but researchers have focused on the following factors: | |||
**[[Gastric]] motor function dysfunction | |||
**[[Visceral]] [[sensitivity]] | |||
**[[Helicobacter pylori infection]] | |||
**[[Psychosocial]] factors | |||
==References== | ==References== | ||
{{Reflist|2}} | {{Reflist|2}} | ||
{{WH}} | |||
{{WS}} | |||
[[Category:Gastroenterology]] | [[Category:Gastroenterology]] | ||
[[Category:Digestive disease symptoms]] | [[Category:Digestive disease symptoms]] | ||
Latest revision as of 21:29, 29 July 2020
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Ajay Gade MD[2]]
Overview
Dyspepsia is broadly classified into ulcer and non-ulcer dyspepsia. The latter is also known as functional dyspepsia.
Classification
Dyspepsia may be broadly classified into two major sub-types:
Ulcer dyspepsia
- Ulcer dyspepsia accounts for 20-30 % of all dyspepsia cases and is caused by peptic ulcer disease and gastroesophageal reflux disease (GERD).
- Structural disease of the gastrointestinal tract exists in ulcer dyspepsia and hence endoscopy is abnormal.
Non-Ulcer dyspepsia
- Non-ulcer dyspepsia, also called functional dyspepsia (FD), is defined by the Rome III criteria as symptoms of epigastric pain or discomfort (found in 89-90% of cases), postprandial fullness (75-88%), and early satiety (50-82%) within the last 3 months with symptom onset at least 6 months earlier.
- Patients should not have any evidence of structural disease and predominant symptoms of gastroesophageal reflux are exclusionary.[1]
- Causes of functional dyspepsia are not clear but researchers have focused on the following factors:
- Gastric motor function dysfunction
- Visceral sensitivity
- Helicobacter pylori infection
- Psychosocial factors