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{{Cerebral venous sinus thrombosis}}
{{Cerebral venous sinus thrombosis}}


There are more than 100 different causes of cerebral venous sinus thrombosis (CVT). Virchow's principles (blood stasis, modification of vascular wall, and blood rheology) should be used to identify all the predisposing conditions for CVT. There are also some genetic and acquired causes leading to CVT. In the cohort of an International study of cerebral vein and dural sinus thrombosis(ISCVT), 34% of patients had thrombophilia while 22% of them had acquired thrombophilia.
There are more than 100 different causes of [[cerebral venous sinus thrombosis]] (CVT). Virchow's principles ([[blood]] stasis, modification of [[vascular]] wall, and [[blood]] [[rheology]]) should be used to identify all the predisposing [[conditions]] for CVT. There are also some [[genetic]] and acquired [[causes]] leading to CVT. In the [[cohort]] of an International study of [[cerebral]] [[vein]] and dural sinus [[thrombosis]](ISCVT), 34% of [[patients]] had [[thrombophilia]] while 22% of them had acquired [[thrombophilia]].
[[Genetic]] prothrombotic causes:
- [[Antithrombin]] [[deficiency]]
- [[Protein C]] and S [[deficiency]]
- Factor v Leiden [[mutation]]
- [[Resistance]] to activated [[protein C]]


== References ==
== References ==

Revision as of 13:18, 15 July 2021

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There are more than 100 different causes of cerebral venous sinus thrombosis (CVT). Virchow's principles (blood stasis, modification of vascular wall, and blood rheology) should be used to identify all the predisposing conditions for CVT. There are also some genetic and acquired causes leading to CVT. In the cohort of an International study of cerebral vein and dural sinus thrombosis(ISCVT), 34% of patients had thrombophilia while 22% of them had acquired thrombophilia. Genetic prothrombotic causes: - Antithrombin deficiency - Protein C and S deficiency - Factor v Leiden mutation - Resistance to activated protein C

References


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