Brugada syndrome differential diagnosis: Difference between revisions
Jump to navigation
Jump to search
| Line 33: | Line 33: | ||
* Other normal variants (particularly in males) | * Other normal variants (particularly in males) | ||
Duchenne muscular dystrophy | |||
thiamin deficiency | |||
hyperkalemia | |||
hypercalcemia | |||
arrhythmogenic right ventricular dysplasia/cardiomyopathy, | |||
pectus excavatum | |||
hypothermia | |||
and mechanical compression of the right ventricular outflow tract (RVOT) as occurs in mediastinal tumor or hemopericardium | |||
Of note, a Brugada-like ECG can occasionally appear for a brief period or for a period of several hours after direct-current cardioversion; it is not known whether these patients are gene carriers for Brugada syndrome. | Of note, a Brugada-like ECG can occasionally appear for a brief period or for a period of several hours after direct-current cardioversion; it is not known whether these patients are gene carriers for Brugada syndrome. | ||
==References== | ==References== | ||
Revision as of 04:03, 14 October 2012
|
Brugada syndrome Microchapters |
|
Diagnosis |
|---|
|
Treatment |
|
Case Studies |
|
Brugada syndrome differential diagnosis On the Web |
|
American Roentgen Ray Society Images of Brugada syndrome differential diagnosis |
|
Risk calculators and risk factors for Brugada syndrome differential diagnosis |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Abnormalities that can lead to ST-segment elevation in the right precordial leads include the following:[1]
- Acute myocardial ischemia or infarction
- Acute myocarditis
- Acute pericarditis
- Acute pulmonary thromboemboli
- Arrhythmogenic right ventricular dysplasia / cardiomyopathy (ARVD/C)
- Cocaine intoxication
- Coronary spasm
- Dissecting aortic aneurysm
- Duchenne muscular dystrophy
- Early repolarization
- Friedreich ataxia
- Heterocyclic antidepressant overdose
- Hypercalcemia
- Hyperkalemia
- Hypothermia, causing Osborn wave in ECGs and sometimes resembling Brugada syndrome
- Left ventricular hypertrophy
- Prinzmetal's angina[2]
- Mediastinal tumor compressing the right ventricular outflow tract (RVOT)
- Right (atypical) or left bundle-branch block
- Right ventricular infarction
- Right ventricular ischemia
- Thiamine deficiency
- Various central and autonomic nervous system abnormalities
- Other conditions that can lead to ST-segment elevation in the right precordial leads
- Early repolarization syndrome
- Other normal variants (particularly in males)
Duchenne muscular dystrophy thiamin deficiency hyperkalemia hypercalcemia arrhythmogenic right ventricular dysplasia/cardiomyopathy, pectus excavatum hypothermia and mechanical compression of the right ventricular outflow tract (RVOT) as occurs in mediastinal tumor or hemopericardium
Of note, a Brugada-like ECG can occasionally appear for a brief period or for a period of several hours after direct-current cardioversion; it is not known whether these patients are gene carriers for Brugada syndrome.
References
- ↑ Takehara N, Makita N, Kawabe J, Sato N, Kawamura Y, Kitabatake A, Kikuchi K (2004). "A cardiac sodium channel mutation identified in Brugada syndrome associated with atrial standstill". Journal of Internal Medicine. 255 (1): 137–42. PMID 14687250. Retrieved 2012-10-13. Unknown parameter
|month=ignored (help) - ↑ Wang K, Asinger RW, Marriott HJ (2003). "ST-segment elevation in conditions other than acute myocardial infarction". The New England Journal of Medicine. 349 (22): 2128–35. doi:10.1056/NEJMra022580. PMID 14645641. Retrieved 2012-10-13. Unknown parameter
|month=ignored (help)