Amyloidosis electrocardiogram: Difference between revisions

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==Overview==
==Overview==
[[Electrocardiogram]] is particularly useful for cardiac amyloidosis. Findings on [[electrocardiogram]] include low voltage [[QRS complexes]], left and right ventricular [[Hypertrophy (medical)|hypertrophy]], [[Left atrium|left atrial]] abnormalities, pathological [[Q wave|Q waves]], and [[Atrioventricular block|AV block]].


==Electrocardiogram==
==Electrocardiogram==
* Echocardiographic features in cardiac amyloidosis include:
[[Electrocardiogram]] is particularly useful for cardiac amyloidosis. Findings on an [[electrocardiogram]] include:
** Wall thickening
*Low voltage QRS complexes<ref name="pmid22058156">{{cite journal| author=Guan J, Mishra S, Falk RH, Liao R| title=Current perspectives on cardiac amyloidosis. | journal=Am J Physiol Heart Circ Physiol | year= 2012 | volume= 302 | issue= 3 | pages= H544-52 | pmid=22058156 | doi=10.1152/ajpheart.00815.2011 | pmc=3353775 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=22058156  }} </ref>: Low voltages are present due to infiltrative heart disease and the reduced electrical conduction through the cardiac muscle.
** Diastolic relaxation abnormalities
*[[Left ventricle|Left ventricular]] [[Hypertrophy (medical)|hypertrophy]]<ref name="pmid25743445">{{cite journal| author=Damy T, Judge DP, Kristen AV, Berthet K, Li H, Aarts J| title=Cardiac findings and events observed in an open-label clinical trial of tafamidis in patients with non-Val30Met and non-Val122Ile hereditary transthyretin amyloidosis. | journal=J Cardiovasc Transl Res | year= 2015 | volume= 8 | issue= 2 | pages= 117-27 | pmid=25743445 | doi=10.1007/s12265-015-9613-9 | pmc=4382536 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=25743445  }} </ref>
** Reduced systolic function
*[[Right ventricle|Right ventricular]] hypertrophy<ref name="pmid25743445">{{cite journal| author=Damy T, Judge DP, Kristen AV, Berthet K, Li H, Aarts J| title=Cardiac findings and events observed in an open-label clinical trial of tafamidis in patients with non-Val30Met and non-Val122Ile hereditary transthyretin amyloidosis. | journal=J Cardiovasc Transl Res | year= 2015 | volume= 8 | issue= 2 | pages= 117-27 | pmid=25743445 | doi=10.1007/s12265-015-9613-9 | pmc=4382536 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=25743445  }} </ref>
*[[Left atrium|Left atrial]] abnormalities
*Pathological [[Q waves]]<ref name="pmid25743445">{{cite journal| author=Damy T, Judge DP, Kristen AV, Berthet K, Li H, Aarts J| title=Cardiac findings and events observed in an open-label clinical trial of tafamidis in patients with non-Val30Met and non-Val122Ile hereditary transthyretin amyloidosis. | journal=J Cardiovasc Transl Res | year= 2015 | volume= 8 | issue= 2 | pages= 117-27 | pmid=25743445 | doi=10.1007/s12265-015-9613-9 | pmc=4382536 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=25743445  }} </ref>
*[[Atrioventricular block]]: [[Atrioventricular block|AV block]] occur due to deposition of amyloid fibrils in the conduction [[Tissue (biology)|tissue]].
**[[First degree AV block|First-degree AV block]]
**[[Second degree AV block|Second-degree AV block]]
**[[Third degree AV block|Third-degree AV block]]


== References ==
== References ==

Latest revision as of 01:15, 26 October 2019

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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Shyam Patel [2]

Overview

Electrocardiogram is particularly useful for cardiac amyloidosis. Findings on electrocardiogram include low voltage QRS complexes, left and right ventricular hypertrophy, left atrial abnormalities, pathological Q waves, and AV block.

Electrocardiogram

Electrocardiogram is particularly useful for cardiac amyloidosis. Findings on an electrocardiogram include:

References

  1. Guan J, Mishra S, Falk RH, Liao R (2012). "Current perspectives on cardiac amyloidosis". Am J Physiol Heart Circ Physiol. 302 (3): H544–52. doi:10.1152/ajpheart.00815.2011. PMC 3353775. PMID 22058156.
  2. 2.0 2.1 2.2 Damy T, Judge DP, Kristen AV, Berthet K, Li H, Aarts J (2015). "Cardiac findings and events observed in an open-label clinical trial of tafamidis in patients with non-Val30Met and non-Val122Ile hereditary transthyretin amyloidosis". J Cardiovasc Transl Res. 8 (2): 117–27. doi:10.1007/s12265-015-9613-9. PMC 4382536. PMID 25743445.

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