Amnesia pathophysiology: Difference between revisions
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==Pathophysiology== | ==Pathophysiology== | ||
===Physiology=== | ===Physiology=== | ||
[[Memory]] is the stored information in the [[brain]] from the past. According to Richard Semon (1904) experiences cause some changes in the [[neurons]] and these changes are referred to as ''engram'' and they form memory of the particular experience. Reactivation of these [[neurons]] occur when [[patient]] tries to recall those [[memories]].<ref>Semon R. (1904). Die mneme [The mneme]. Edited by W. Engelmann. Leipzig</ref> | [[Memory]] is the stored information in the [[brain]] from the past. According to Richard Semon (1904) experiences cause some structural and functional changes in the [[neurons]] and these changes are referred to as ''engram'' and they form memory of the particular experience. Reactivation of these [[neurons]] occur when [[patient]] tries to recall those [[memories]].<ref>Semon R. (1904). Die mneme [The mneme]. Edited by W. Engelmann. Leipzig</ref> Memory is divided in two groups depending on the duration: | ||
*Short-term [[memory]] are for short period of time and use existing [[neuronal]] network. | |||
*Long term [[memory]] are long lasting and are formed by structural/functional changes in neuronal network. | |||
===Pathogenesis=== | ===Pathogenesis=== | ||
Revision as of 00:52, 9 March 2021
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Amnesia Microchapters |
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Amnesia pathophysiology On the Web |
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American Roentgen Ray Society Images of Amnesia pathophysiology |
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Risk calculators and risk factors for Amnesia pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Zehra Malik, M.B.B.S[2]
Overview
The exact pathogenesis of [disease name] is not fully understood.
OR
It is thought that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
OR
[Pathogen name] is usually transmitted via the [transmission route] route to the human host.
OR
Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
OR
[Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
OR
The progression to [disease name] usually involves the [molecular pathway].
OR
The pathophysiology of [disease/malignancy] depends on the histological subtype.
Pathophysiology
Physiology
Memory is the stored information in the brain from the past. According to Richard Semon (1904) experiences cause some structural and functional changes in the neurons and these changes are referred to as engram and they form memory of the particular experience. Reactivation of these neurons occur when patient tries to recall those memories.[1] Memory is divided in two groups depending on the duration:
- Short-term memory are for short period of time and use existing neuronal network.
- Long term memory are long lasting and are formed by structural/functional changes in neuronal network.
Pathogenesis
- The exact pathogenesis of [disease name] is not completely understood.
OR
- It is understood that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
- [Pathogen name] is usually transmitted via the [transmission route] route to the human host.
- Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
- [Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
- The progression to [disease name] usually involves the [molecular pathway].
- The pathophysiology of [disease/malignancy] depends on the histological subtype.
Genetics
[Disease name] is transmitted in [mode of genetic transmission] pattern.
OR
Genes involved in the pathogenesis of [disease name] include:
- [Gene1]
- [Gene2]
- [Gene3]
OR
The development of [disease name] is the result of multiple genetic mutations such as:
- [Mutation 1]
- [Mutation 2]
- [Mutation 3]
Associated Conditions
Conditions associated with [disease name] include:
- [Condition 1]
- [Condition 2]
- [Condition 3]
Gross Pathology
On gross pathology, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].
Microscopic Pathology
On microscopic histopathological analysis, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].
References
- ↑ Semon R. (1904). Die mneme [The mneme]. Edited by W. Engelmann. Leipzig