Amaurosis fugax causes: Difference between revisions

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*Carotid occlusive disease
*Carotid occlusive disease


*[[Carotid stenosis]]<ref name="pmid22783748">{{cite journal |author=Rozegnał-Madej A, Bielecka E, Swiech-Zubilewicz A, Zarnowski T, Karakuła W, Zubilewicz T |title=[Ophthalmological complications associated with clinically significant carotid stenosis] |language=Polish |journal=Klin Oczna |volume=114 |issue=1 |pages=57–62 |year=2012 |pmid=22783748 |doi= |url=}}</ref>
*[[Carotid stenosis]]<ref name="pmid22783748">{{cite journal |author=Rozegnał-Madej A, Bielecka E, Swiech-Zubilewicz A, Zarnowski T, Karakuła W, Zubilewicz T |title=[Ophthalmological complications associated with clinically significant carotid stenosis] |language=Polish |journal=Klin Oczna |volume=114 |issue=1 |pages=57–62 |year=2012 |pmid=22783748 |doi= |url=}}</ref><ref name="pmid23198268">{{cite journal |author=Tomaschütz L, Dos Santos M, Schill J, Palm F, Grau A |title=Recurrent amaurosis fugax in a patient after stanford type a dissection depending on blood pressure and haemoglobin level |journal=Case Rep Vasc Med |volume=2012 |issue= |pages=254204 |year=2012 |pmid=23198268 |pmc=3502825 |doi=10.1155/2012/254204 |url=}}</ref>


*[[Central retinal vein occlusion]]<ref name="pmid22969282">{{cite journal |author=Glueck CJ, Hutchins RK, Jurantee J, Khan Z, Wang P |title=Thrombophilia and retinal vascular occlusion |journal=Clin Ophthalmol |volume=6 |issue= |pages=1377–84 |year=2012 |pmid=22969282 |pmc=3437951 |doi=10.2147/OPTH.S34627 |url=}}</ref>
*[[Central retinal vein occlusion]]<ref name="pmid22969282">{{cite journal |author=Glueck CJ, Hutchins RK, Jurantee J, Khan Z, Wang P |title=Thrombophilia and retinal vascular occlusion |journal=Clin Ophthalmol |volume=6 |issue= |pages=1377–84 |year=2012 |pmid=22969282 |pmc=3437951 |doi=10.2147/OPTH.S34627 |url=}}</ref>


*Cerebral [[transient ischemic attacks]] (TIAs)<ref name="pmid4083852">{{cite journal |author=Hurwitz BJ, Heyman A, Wilkinson WE, Haynes CS, Utley CM |title=Comparison of amaurosis fugax and transient cerebral ischemia: a prospective clinical and arteriographic study |journal=Ann. Neurol. |volume=18 |issue=6 |pages=698–704 |year=1985 |month=December |pmid=4083852 |doi=10.1002/ana.410180612 |url=}}</ref><ref name="pmid22783748">{{cite journal |author=Rozegnał-Madej A, Bielecka E, Swiech-Zubilewicz A, Zarnowski T, Karakuła W, Zubilewicz T |title=[Ophthalmological complications associated with clinically significant carotid stenosis] |language=Polish |journal=Klin Oczna |volume=114 |issue=1 |pages=57–62 |year=2012 |pmid=22783748 |doi= |url=}}</ref>
*Cerebral [[transient ischemic attacks]] (TIAs)<ref name="pmid4083852">{{cite journal |author=Hurwitz BJ, Heyman A, Wilkinson WE, Haynes CS, Utley CM |title=Comparison of amaurosis fugax and transient cerebral ischemia: a prospective clinical and arteriographic study |journal=Ann. Neurol. |volume=18 |issue=6 |pages=698–704 |year=1985 |month=December |pmid=4083852 |doi=10.1002/ana.410180612 |url=}}</ref><ref name="pmid22783748">{{cite journal |author=Rozegnał-Madej A, Bielecka E, Swiech-Zubilewicz A, Zarnowski T, Karakuła W, Zubilewicz T |title=[Ophthalmological complications associated with clinically significant carotid stenosis] |language=Polish |journal=Klin Oczna |volume=114 |issue=1 |pages=57–62 |year=2012 |pmid=22783748 |doi= |url=}}</ref><ref name="pmid23198268">{{cite journal |author=Tomaschütz L, Dos Santos M, Schill J, Palm F, Grau A |title=Recurrent amaurosis fugax in a patient after stanford type a dissection depending on blood pressure and haemoglobin level |journal=Case Rep Vasc Med |volume=2012 |issue= |pages=254204 |year=2012 |pmid=23198268 |pmc=3502825 |doi=10.1155/2012/254204 |url=}}</ref>


*Chiasm compression
*Chiasm compression
Line 73: Line 73:
* Hypofibrinolysis
* Hypofibrinolysis


*[[Hypoperfusion]] (from [[cardiac failure]])
*[[Hypoperfusion]] (from [[cardiac failure]])<ref name="pmid23198268">{{cite journal |author=Tomaschütz L, Dos Santos M, Schill J, Palm F, Grau A |title=Recurrent amaurosis fugax in a patient after stanford type a dissection depending on blood pressure and haemoglobin level |journal=Case Rep Vasc Med |volume=2012 |issue= |pages=254204 |year=2012 |pmid=23198268 |pmc=3502825 |doi=10.1155/2012/254204 |url=}}</ref>


*[[Hypotension]]
*[[Hypotension]]<ref name="pmid23198268">{{cite journal |author=Tomaschütz L, Dos Santos M, Schill J, Palm F, Grau A |title=Recurrent amaurosis fugax in a patient after stanford type a dissection depending on blood pressure and haemoglobin level |journal=Case Rep Vasc Med |volume=2012 |issue= |pages=254204 |year=2012 |pmid=23198268 |pmc=3502825 |doi=10.1155/2012/254204 |url=}}</ref>


*[[Hypovolemia]]
*[[Hypovolemia]]

Revision as of 20:38, 27 February 2013

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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor-In-Chief: Marcelo R. Zacarkim, M.D. [2]

Overview

Amaurosis fugax is a symptom of carotid artery disease. It occurs when a piece of plaque in a carotid artery breaks off and travels to the retinal artery in the eye. The carotid arteries provide the main blood supply to the brain. They are located on each side of the neck under the jaw. Plaque is a hard substance that forms when fat, cholesterol, and other substances build up in the walls of arteries. Pieces of plaque can block blood flow. In people with amaurosis fugax, vision loss continues as long as the blood supply to the retinal artery is blocked. Atherosclerosis of the arteries in the neck is the main risk factor for this condition. Risk factors for atherosclerosis include heart disease, high cholesterol, smoking, diabetes, and high blood pressure.

Causes in Alphabetical Order

  • Blood coagulation disorders
  • Blood viscosity
  • Cardiac emboli (valve, mural thrombi, intracardiac tumor)
  • Carotid occlusive disease
  • Chiasm compression
  • Decreased blood flow (ischemia) to the retina
  • Disease susceptibility
  • Distal internal carotid artery atheroembolism
  • Extracranial arterial occlusive disease
  • Ipsilateral carotid disease[3]
  • Irregular ulcerated lesion
  • Neurologic disorder
  • Nonvascular ophthalmic disorder
  • Obesity
  • Sudden vision changes


References

  1. 1.0 1.1 1.2 1.3 1.4 1.5 1.6 1.7 Rozegnał-Madej A, Bielecka E, Swiech-Zubilewicz A, Zarnowski T, Karakuła W, Zubilewicz T (2012). "[Ophthalmological complications associated with clinically significant carotid stenosis]". Klin Oczna (in Polish). 114 (1): 57–62. PMID 22783748.
  2. 2.0 2.1 2.2 Alba MA, Mena-Madrazo JA, Reyes E, Flores-Suárez LF (2012). "Giant cell arteritis in Mexican patients". J Clin Rheumatol. 18 (1): 1–7. doi:10.1097/RHU.0b013e31823e2e35. PMID 22157266. Unknown parameter |month= ignored (help)
  3. 3.0 3.1 Huwez F, Umasankar U, Casswell E, Menon J, Gadi N (2011). "Sudden loss of vision in a patient with significant ipsilateral internal carotid disease". BMJ Case Rep. 2011. doi:10.1136/bcr.08.2011.4725. PMID 22669766.
  4. 4.0 4.1 4.2 4.3 Tomaschütz L, Dos Santos M, Schill J, Palm F, Grau A (2012). "Recurrent amaurosis fugax in a patient after stanford type a dissection depending on blood pressure and haemoglobin level". Case Rep Vasc Med. 2012: 254204. doi:10.1155/2012/254204. PMC 3502825. PMID 23198268.
  5. 5.0 5.1 Glueck CJ, Hutchins RK, Jurantee J, Khan Z, Wang P (2012). "Thrombophilia and retinal vascular occlusion". Clin Ophthalmol. 6: 1377–84. doi:10.2147/OPTH.S34627. PMC 3437951. PMID 22969282.
  6. Hurwitz BJ, Heyman A, Wilkinson WE, Haynes CS, Utley CM (1985). "Comparison of amaurosis fugax and transient cerebral ischemia: a prospective clinical and arteriographic study". Ann. Neurol. 18 (6): 698–704. doi:10.1002/ana.410180612. PMID 4083852. Unknown parameter |month= ignored (help)
  7. Hayreh SS, Podhajsky PA, Zimmerman B (1998). "Ocular manifestations of giant cell arteritis". Am. J. Ophthalmol. 125 (4): 509–20. PMID 9559737. Unknown parameter |month= ignored (help)
  8. Hayreh SS, Podhajsky PA, Zimmerman B (1998). "Occult giant cell arteritis: ocular manifestations". Am. J. Ophthalmol. 125 (4): 521–6. PMID 9559738. Unknown parameter |month= ignored (help)
  9. 9.0 9.1 Khan AO (2006). "Severe psychogenic visual loss in a girl with siblings blinded from congenital glaucoma". J AAPOS. 10 (4): 373–4. doi:10.1016/j.jaapos.2006.01.216. PMID 16935241. Unknown parameter |month= ignored (help)
  10. Saric M, Kronzon I (2011). "Cholesterol embolization syndrome". Curr. Opin. Cardiol. 26 (6): 472–9. doi:10.1097/HCO.0b013e32834b7fdd. PMID 21993354. Unknown parameter |month= ignored (help)
  11. Lord RS (1990). "Transient monocular blindness". Aust N Z J Ophthalmol. 18 (3): 299–305. PMID 2261177. Unknown parameter |month= ignored (help)
  12. Mead GE, Lewis SC, Wardlaw JM, Dennis MS (2002). "Comparison of risk factors in patients with transient and prolonged eye and brain ischemic syndromes". Stroke. 33 (10): 2383–90. PMID 12364725. Unknown parameter |month= ignored (help)
  13. Wang JJ, Cugati S, Knudtson MD; et al. (2006). "Retinal arteriolar emboli and long-term mortality: pooled data analysis from two older populations". Stroke. 37 (7): 1833–6. doi:10.1161/01.STR.0000226929.23297.75. PMID 16741179. Unknown parameter |month= ignored (help)
  14. Cugati S, Wang JJ, Rochtchina E, Mitchell P (2006). "Ten-year incidence of retinal emboli in an older population". Stroke. 37 (3): 908–10. doi:10.1161/01.STR.0000204118.87477.46. PMID 16439697. Unknown parameter |month= ignored (help)