Haff disease: Difference between revisions
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===Physical factors=== | ===Physical factors=== | ||
*Trauma | *[[Physical trauma|Trauma]] | ||
*Excessive muscular activity | *Excessive muscular activity | ||
*Status epilepticus | *[[Status epilepticus]] | ||
*High-grade fever | *High-grade [[fever]] | ||
*Heat stroke | *[[Heat stroke]] | ||
*Electric shock | *[[Electric shock]] | ||
===Non-physical factors=== | ===Non-physical factors=== | ||
*Medication (e.g., anti-retroviral, anti-histamine, anti-psychotics, antidepressants, and statins) | *Medication (e.g., [[AIDS antiretroviral drugs|anti-retroviral]], [[anti-histamine]], [[Antipsychotics|anti-psychotics]], [[antidepressants]], and [[statins]]) | ||
*Infection (bacterial, and viral) | *[[Infection]] (bacterial, and viral) | ||
*Drugs (e.g., alcohol, heroin, cocaine, methamphetamine, ecstasy, and LSD) | *Drugs (e.g., [[alcohol]], [[heroin]], [[cocaine]], [[methamphetamine]], [[Ecstasy (drug)|ecstasy]], and [[Lysergic acid diethylamide|LSD]]) | ||
*Electrolyte abnormalities (e.g., hypokalemia, hypophosphatemia, hyponatremia, and hypernatremia) | *Electrolyte abnormalities (e.g., [[hypokalemia]], [[hypophosphatemia]], [[hyponatremia]], and [[hypernatremia]]) | ||
===Genetic factors=== | ===Genetic factors=== | ||
*Autoimmune diseases (e.g., polymyositis and dermatomyositis) | *Autoimmune diseases (e.g., [[polymyositis and dermatomyositis]]) | ||
*Metabolic diseases (e.g., diabetes, thyroid dysfunction, primary hyperaldosteronism, primary adrenal insufficiency, central diabetes insipidus, postpartum hypernatremia, and pituitary dysfunction). | *Metabolic diseases (e.g., [[Diabetes mellitus|diabetes]], [[Thyroid disease|thyroid dysfunction]], [[primary hyperaldosteronism]], [[primary adrenal insufficiency]], [[central diabetes insipidus]], postpartum hypernatremia, and pituitary dysfunction). | ||
*Inherited conditions (e.g., glycogen storage disorders, fatty acid oxidation disorders, mitochondrial myopathies, muscular dystrophies, and RYR1- related myopathies) | *Inherited conditions (e.g., [[Glycogen storage disease|glycogen storage disorders]], [[Fatty acid oxidation disorder|fatty acid oxidation disorders]], [[Mitochondrial myopathy|mitochondrial myopathies]], [[Muscular dystrophy|muscular dystrophies]], and RYR1- related myopathies) | ||
==[[Haff disease epidemiology and demographics|Epidemiology and Demographics]]== | ==[[Haff disease epidemiology and demographics|Epidemiology and Demographics]]== | ||
Revision as of 03:11, 26 March 2022
| Haff disease | |
| Haff disease was first described in the location of Königsberg/Frisches Haff[1] | |
| ICD-9 | 985.1 |
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| DiseasesDB | 33568 |
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Haff disease Microchapters |
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Diagnosis |
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Treatment |
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Case Studies |
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Haff disease On the Web |
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American Roentgen Ray Society Images of Haff disease |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-In-Chief: M. Hassan, M.B.B.S
Overview
Historical Perspective
Pathophysiology
Causes
Differentiating Haff disease from other Diseases
Haff disease is known to cause rhabdomyolysis, and therefore, it should be differentiated from other causes of rhabdomyolysis. There are numerous etiologies of rhabdomyolysis and they may be categorized based on the physical, non-physical and genetic factors.
Physical factors
- Trauma
- Excessive muscular activity
- Status epilepticus
- High-grade fever
- Heat stroke
- Electric shock
Non-physical factors
- Medication (e.g., anti-retroviral, anti-histamine, anti-psychotics, antidepressants, and statins)
- Infection (bacterial, and viral)
- Drugs (e.g., alcohol, heroin, cocaine, methamphetamine, ecstasy, and LSD)
- Electrolyte abnormalities (e.g., hypokalemia, hypophosphatemia, hyponatremia, and hypernatremia)
Genetic factors
- Autoimmune diseases (e.g., polymyositis and dermatomyositis)
- Metabolic diseases (e.g., diabetes, thyroid dysfunction, primary hyperaldosteronism, primary adrenal insufficiency, central diabetes insipidus, postpartum hypernatremia, and pituitary dysfunction).
- Inherited conditions (e.g., glycogen storage disorders, fatty acid oxidation disorders, mitochondrial myopathies, muscular dystrophies, and RYR1- related myopathies)
Epidemiology and Demographics
Risk Factors
Natural History, Complications and Prognosis
Diagnosis
History and Symptoms | Physical Examination | Laboratory Findings | Electrocardiogram | Ultrasound | Other Diagnostic Studies
Treatment
Medical Therapy | Surgery | Primary Prevention | Cost-Effectiveness of Therapy | Future or Investigational Therapies
References
- ↑ Template:Dorlands
- ↑ Nance JR, Mammen AL (2015). "Diagnostic evaluation of rhabdomyolysis". Muscle Nerve. 51 (6): 793–810. doi:10.1002/mus.24606. PMC 4437836. PMID 25678154.