Dyslipidemia resident survival guide: Difference between revisions
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==Classification== | ==Classification== | ||
==Causes== | ==Causes== | ||
Secondary causes of dyslipidemia may cause either an increase in total-cholesterol/low density lipoprotein-cholesterol (LDL-C) or an increase in total triglycerides & very low density lipoprotein cholesterol (VLDL-C) | |||
===Increase in Total Cholesterol and LDL-C=== | |||
*Hypothyroidism | |||
*Nephrosis | |||
*Dysgammaglobulinemia (systemic lupus erythematosus, multiple myeloma) | |||
*Cholestatic hepatic diseases due to abnormal lipoproteins (e.g. primary biliary cirrhosis) | |||
*Administration of protease inhibitors (treatment for HIV infection) | |||
*Administration of progestin or anabolic steroids | |||
===Increase in Total Triglycerides and VLDL-C=== | |||
*Chronic kidney disease | |||
*Type 2 diabetes mellitus | |||
*Obesity | |||
*Excessive alcohol intake | |||
*Hypothyroidism | |||
*Administration of anti-hypertensive therapy (thiazide diuretics or B-blockers) | |||
*Administration of corticosteroids | |||
*Severe stress that increases endogenous corticosteroid concentration | |||
*Elevated concentrations of estrogen (administration of oral (not transdermal) estrogen therapy, oral contraceptives, or pregnancy) | |||
*Administration of protease inhibitors (treatment for HIV infection) | |||
==Complete Diagnostic Approach== | ==Complete Diagnostic Approach== | ||
==Treatment== | ==Treatment== | ||
Revision as of 14:28, 20 April 2015
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief:
Overview
Classification
Causes
Secondary causes of dyslipidemia may cause either an increase in total-cholesterol/low density lipoprotein-cholesterol (LDL-C) or an increase in total triglycerides & very low density lipoprotein cholesterol (VLDL-C)
Increase in Total Cholesterol and LDL-C
- Hypothyroidism
- Nephrosis
- Dysgammaglobulinemia (systemic lupus erythematosus, multiple myeloma)
- Cholestatic hepatic diseases due to abnormal lipoproteins (e.g. primary biliary cirrhosis)
- Administration of protease inhibitors (treatment for HIV infection)
- Administration of progestin or anabolic steroids
Increase in Total Triglycerides and VLDL-C
- Chronic kidney disease
- Type 2 diabetes mellitus
- Obesity
- Excessive alcohol intake
- Hypothyroidism
- Administration of anti-hypertensive therapy (thiazide diuretics or B-blockers)
- Administration of corticosteroids
- Severe stress that increases endogenous corticosteroid concentration
- Elevated concentrations of estrogen (administration of oral (not transdermal) estrogen therapy, oral contraceptives, or pregnancy)
- Administration of protease inhibitors (treatment for HIV infection)