Posterior reversible encephalopathy syndrome

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Posterior reversible encephalopathy syndrome
DiseasesDB 10460

Posterior reversible encephalopathy syndrome (PRES) is a syndrome characterised by headache, confusion, seizures and visual loss associated with imaging findings of bilateral cortical and subcortical brain edema. Common precipitating factors are abrupt elevations of blood pressure, renal decompensation, fluid retention, and immunosuppressive therapy. It was first described in 1996.[1]

It is also known as reversible posterior Leukoencephalopathy syndrome [RPLS]. it occurs due to elevation in Blood pressure. The condition is reversed by control of Blood pressure and withdrawal of the drug producing the symptoms.

MRI Images with Axial Flare

Life Threatening Causes

Common Causes

Causes by Organ System

Cardiovascular No underlying causes
Chemical/Poisoning No underlying causes
Dental No underlying causes
Dermatologic No underlying causes
Drug Side Effect Axitinib, Lenvatinib, Sorafenib
Ear Nose Throat No underlying causes
Endocrine No underlying causes
Environmental No underlying causes
Gastroenterologic No underlying causes
Genetic No underlying causes
Hematologic No underlying causes
Iatrogenic No underlying causes
Infectious Disease No underlying causes
Musculoskeletal/Orthopedic No underlying causes
Neurologic No underlying causes
Nutritional/Metabolic No underlying causes
Obstetric/Gynecologic No underlying causes
Oncologic No underlying causes
Ophthalmologic No underlying causes
Overdose/Toxicity No underlying causes
Psychiatric No underlying causes
Pulmonary No underlying causes
Renal/Electrolyte No underlying causes
Rheumatology/Immunology/Allergy No underlying causes
Sexual No underlying causes
Trauma No underlying causes
Urologic No underlying causes
Miscellaneous No underlying causes

Causes in Alphabetical Order


References

  1. Hinchey J, Chaves C, Appignani B, Breen J, Pao L, Wang A, Pessin M, Lamy C, Mas J, Caplan L (1996). "A reversible posterior leukoencephalopathy syndrome". N Engl J Med. 334 (8): 494–500. PMID 8559202.

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